The secondary FLT3-ITD F691L mutation induces resistance to AC220 in FLT3-ITD+ AML but retains in vitro sensitivity to PKC412 and Sunitinib
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The secondary FLT3-ITD F691L mutation induces resistance to AC220 in FLT3-ITD+ AML but retains in vitro sensitivity to PKC412 and Sunitinib. / Albers, C; Leischner, H; Verbeek, M; Yu, C; Illert, A L; Peschel, C; von Bubnoff, N; Duyster, J.
In: LEUKEMIA, Vol. 27, No. 6, 06.2013, p. 1416-8.Research output: SCORING: Contribution to journal › SCORING: Journal article › Research › peer-review
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TY - JOUR
T1 - The secondary FLT3-ITD F691L mutation induces resistance to AC220 in FLT3-ITD+ AML but retains in vitro sensitivity to PKC412 and Sunitinib
AU - Albers, C
AU - Leischner, H
AU - Verbeek, M
AU - Yu, C
AU - Illert, A L
AU - Peschel, C
AU - von Bubnoff, N
AU - Duyster, J
PY - 2013/6
Y1 - 2013/6
KW - Antineoplastic Agents
KW - Benzothiazoles
KW - Drug Resistance, Neoplasm
KW - Female
KW - Humans
KW - Indoles
KW - Leukemia, Myeloid, Acute
KW - Middle Aged
KW - Mutation
KW - Phenylurea Compounds
KW - Pyrroles
KW - Staurosporine
KW - fms-Like Tyrosine Kinase 3
KW - Case Reports
KW - Letter
U2 - 10.1038/leu.2013.14
DO - 10.1038/leu.2013.14
M3 - SCORING: Journal article
C2 - 23392356
VL - 27
SP - 1416
EP - 1418
JO - LEUKEMIA
JF - LEUKEMIA
SN - 0887-6924
IS - 6
ER -