Research ExplorerScientific StaffCorinna Albers-Leischner

Dr. ID: 6016847

Corinna Albers-Leischner

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Affiliations

Publications

Mobocertinib in Patients with EGFR Exon 20 Insertion-Positive Non-Small Cell Lung Cancer (MOON): An International Real-World Safety and Efficacy Analysis

Research output: SCORING: Contribution to journal › SCORING: Journal article › Research › peer-review

Treatment outcome of atypical EGFR mutations in the German National Network Genomic Medicine Lung Cancer (nNGM)

Research output: SCORING: Contribution to journal › SCORING: Journal article › Research › peer-review

NIPA (Nuclear Interaction Partner of ALK) Is Crucial for Effective NPM-ALK Mediated Lymphomagenesis

Research output: SCORING: Contribution to journal › SCORING: Journal article › Research › peer-review

Phosphorylation of Beclin-1 by BCR-ABL suppresses autophagy in chronic myeloid leukemia

Research output: SCORING: Contribution to journal › SCORING: Journal article › Research › peer-review

Grb10 is involved in BCR-ABL-positive leukemia in mice

Research output: SCORING: Contribution to journal › SCORING: Journal article › Research › peer-review

GP130 activation induces myeloma and collaborates with MYC

Research output: SCORING: Contribution to journal › SCORING: Journal article › Research › peer-review

The secondary FLT3-ITD F691L mutation induces resistance to AC220 in FLT3-ITD+ AML but retains in vitro sensitivity to PKC412 and Sunitinib

Research output: SCORING: Contribution to journal › SCORING: Journal article › Research › peer-review

Extracellular signal-regulated kinase 2 (ERK2) mediates phosphorylation and inactivation of nuclear interaction partner of anaplastic lymphoma kinase (NIPA) at G2/M

Research output: SCORING: Contribution to journal › SCORING: Journal article › Research › peer-review

SRC is a signaling mediator in FLT3-ITD- but not in FLT3-TKD-positive AML

Research output: SCORING: Contribution to journal › SCORING: Journal article › Research › peer-review

An RNAi-based system for loss-of-function analysis identifies Raf1 as a crucial mediator of BCR-ABL-driven leukemogenesis

Research output: SCORING: Contribution to journal › SCORING: Journal article › Research › peer-review

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