Research ExplorerPublicationsThe ins-and-outs of podocyte lipid metabolism

The ins-and-outs of podocyte lipid metabolism

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The ins-and-outs of podocyte lipid metabolism. / Meyer-Schwesinger, Catherine.

In: KIDNEY INT, Vol. 98, No. 5, 11.2020, p. 1087-1090.

Research output: SCORING: Contribution to journal › Other (editorial matter etc.) › Research

Harvard

Meyer-Schwesinger, C 2020, 'The ins-and-outs of podocyte lipid metabolism', KIDNEY INT, vol. 98, no. 5, pp. 1087-1090. https://doi.org/10.1016/j.kint.2020.07.008

APA

Meyer-Schwesinger, C. (2020). The ins-and-outs of podocyte lipid metabolism. KIDNEY INT, 98(5), 1087-1090. https://doi.org/10.1016/j.kint.2020.07.008

Vancouver

Meyer-Schwesinger C. The ins-and-outs of podocyte lipid metabolism. KIDNEY INT. 2020 Nov;98(5):1087-1090. https://doi.org/10.1016/j.kint.2020.07.008

Bibtex

@article{570e80dcef4041dbb486aadb7f7c332c,
title = "The ins-and-outs of podocyte lipid metabolism",
abstract = "Obesity and hyperlipidemia are the most prevalent independent risk factors of ESRD, suggesting that lipid accumulation is detrimental to renal function. The origin of lipid accumulation (a common feature in podocyte injury) and its pathophysiological relevance are unknown. This commentary discusses the finding by Liu et al. that deficiency of the endoplasmic reticulum enzyme SOAT1, which metabolizes cholesterol to cholesterol esters, attenuates renal/podocyte injury in murine models of diabetes and Alport's syndrome.",
keywords = "Animals, Diabetes Mellitus/metabolism, Kidney, Lipid Metabolism, Mice, Nephritis, Hereditary/metabolism, Podocytes/metabolism",
author = "Catherine Meyer-Schwesinger",
note = "Editorial Material",
year = "2020",
month = nov,
doi = "10.1016/j.kint.2020.07.008",
language = "English",
volume = "98",
pages = "1087--1090",
journal = "KIDNEY INT",
issn = "0085-2538",
publisher = "NATURE PUBLISHING GROUP",
number = "5",

}

RIS

TY - JOUR

T1 - The ins-and-outs of podocyte lipid metabolism

AU - Meyer-Schwesinger, Catherine

N1 - Editorial Material

PY - 2020/11

Y1 - 2020/11

N2 - Obesity and hyperlipidemia are the most prevalent independent risk factors of ESRD, suggesting that lipid accumulation is detrimental to renal function. The origin of lipid accumulation (a common feature in podocyte injury) and its pathophysiological relevance are unknown. This commentary discusses the finding by Liu et al. that deficiency of the endoplasmic reticulum enzyme SOAT1, which metabolizes cholesterol to cholesterol esters, attenuates renal/podocyte injury in murine models of diabetes and Alport's syndrome.

AB - Obesity and hyperlipidemia are the most prevalent independent risk factors of ESRD, suggesting that lipid accumulation is detrimental to renal function. The origin of lipid accumulation (a common feature in podocyte injury) and its pathophysiological relevance are unknown. This commentary discusses the finding by Liu et al. that deficiency of the endoplasmic reticulum enzyme SOAT1, which metabolizes cholesterol to cholesterol esters, attenuates renal/podocyte injury in murine models of diabetes and Alport's syndrome.

KW - Animals

KW - Diabetes Mellitus/metabolism

KW - Kidney

KW - Lipid Metabolism

KW - Mice

KW - Nephritis, Hereditary/metabolism

KW - Podocytes/metabolism

U2 - 10.1016/j.kint.2020.07.008

DO - 10.1016/j.kint.2020.07.008

M3 - Other (editorial matter etc.)

C2 - 33126971

VL - 98

SP - 1087

EP - 1090

JO - KIDNEY INT

JF - KIDNEY INT

SN - 0085-2538

IS - 5

ER -