The clock genes Period 2 and Cryptochrome 2 differentially balance bone formation.

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The clock genes Period 2 and Cryptochrome 2 differentially balance bone formation. / Maronde, Erik; Schilling, Arndt; Seitz, Sebastian; Schinke, Thorsten; Schmutz, Isabelle; van der Horst, Gijsbertus; Amling, Michael; Albrecht, Urs.

In: PLOS ONE, Vol. 5, No. 7, 7, 2010, p. 11527.

Research output: SCORING: Contribution to journalSCORING: Journal articleResearchpeer-review

Harvard

Maronde, E, Schilling, A, Seitz, S, Schinke, T, Schmutz, I, van der Horst, G, Amling, M & Albrecht, U 2010, 'The clock genes Period 2 and Cryptochrome 2 differentially balance bone formation.', PLOS ONE, vol. 5, no. 7, 7, pp. 11527. https://doi.org/10.1371/journal.pone.0011527

APA

Maronde, E., Schilling, A., Seitz, S., Schinke, T., Schmutz, I., van der Horst, G., Amling, M., & Albrecht, U. (2010). The clock genes Period 2 and Cryptochrome 2 differentially balance bone formation. PLOS ONE, 5(7), 11527. [7]. https://doi.org/10.1371/journal.pone.0011527

Vancouver

Bibtex

@article{1f3e73c615b8431da2c4dd87a0d6954b,
title = "The clock genes Period 2 and Cryptochrome 2 differentially balance bone formation.",
abstract = "Clock genes and their protein products regulate circadian rhythms in mammals but have also been implicated in various physiological processes, including bone formation. Osteoblasts build new mineralized bone whereas osteoclasts degrade it thereby balancing bone formation. To evaluate the contribution of clock components in this process, we investigated mice mutant in clock genes for a bone volume phenotype.",
keywords = "Animals, Mice, Mice, Mutant Strains, Bone Density genetics, Cryptochromes genetics, Osteoblasts cytology, Osteoclasts cytology, Osteogenesis genetics, Period Circadian Proteins genetics, Animals, Mice, Mice, Mutant Strains, Bone Density genetics, Cryptochromes genetics, Osteoblasts cytology, Osteoclasts cytology, Osteogenesis genetics, Period Circadian Proteins genetics",
author = "Erik Maronde and Arndt Schilling and Sebastian Seitz and Thorsten Schinke and Isabelle Schmutz and {van der Horst}, Gijsbertus and Michael Amling and Urs Albrecht",
year = "2010",
doi = "10.1371/journal.pone.0011527",
language = "Deutsch",
volume = "5",
pages = "11527",
journal = "PLOS ONE",
issn = "1932-6203",
publisher = "Public Library of Science",
number = "7",

}

RIS

TY - JOUR

T1 - The clock genes Period 2 and Cryptochrome 2 differentially balance bone formation.

AU - Maronde, Erik

AU - Schilling, Arndt

AU - Seitz, Sebastian

AU - Schinke, Thorsten

AU - Schmutz, Isabelle

AU - van der Horst, Gijsbertus

AU - Amling, Michael

AU - Albrecht, Urs

PY - 2010

Y1 - 2010

N2 - Clock genes and their protein products regulate circadian rhythms in mammals but have also been implicated in various physiological processes, including bone formation. Osteoblasts build new mineralized bone whereas osteoclasts degrade it thereby balancing bone formation. To evaluate the contribution of clock components in this process, we investigated mice mutant in clock genes for a bone volume phenotype.

AB - Clock genes and their protein products regulate circadian rhythms in mammals but have also been implicated in various physiological processes, including bone formation. Osteoblasts build new mineralized bone whereas osteoclasts degrade it thereby balancing bone formation. To evaluate the contribution of clock components in this process, we investigated mice mutant in clock genes for a bone volume phenotype.

KW - Animals

KW - Mice

KW - Mice, Mutant Strains

KW - Bone Density genetics

KW - Cryptochromes genetics

KW - Osteoblasts cytology

KW - Osteoclasts cytology

KW - Osteogenesis genetics

KW - Period Circadian Proteins genetics

KW - Animals

KW - Mice

KW - Mice, Mutant Strains

KW - Bone Density genetics

KW - Cryptochromes genetics

KW - Osteoblasts cytology

KW - Osteoclasts cytology

KW - Osteogenesis genetics

KW - Period Circadian Proteins genetics

U2 - 10.1371/journal.pone.0011527

DO - 10.1371/journal.pone.0011527

M3 - SCORING: Zeitschriftenaufsatz

VL - 5

SP - 11527

JO - PLOS ONE

JF - PLOS ONE

SN - 1932-6203

IS - 7

M1 - 7

ER -