Loss of the coxsackie and adenovirus receptor contributes to gastric cancer progression.

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Loss of the coxsackie and adenovirus receptor contributes to gastric cancer progression. / Anders, Mario; Vieth, M; Röcken, C; Ebert, M; Pross, M; Gretschel, S; Schlag, P M; Wiedenmann, B; Kemmner, W; Höcker, M.

In: BRIT J CANCER, Vol. 100, No. 2, 2, 2009, p. 352-359.

Research output: SCORING: Contribution to journalSCORING: Journal articleResearchpeer-review

Harvard

Anders, M, Vieth, M, Röcken, C, Ebert, M, Pross, M, Gretschel, S, Schlag, PM, Wiedenmann, B, Kemmner, W & Höcker, M 2009, 'Loss of the coxsackie and adenovirus receptor contributes to gastric cancer progression.', BRIT J CANCER, vol. 100, no. 2, 2, pp. 352-359. <http://www.ncbi.nlm.nih.gov/pubmed/19142187?dopt=Citation>

APA

Anders, M., Vieth, M., Röcken, C., Ebert, M., Pross, M., Gretschel, S., Schlag, P. M., Wiedenmann, B., Kemmner, W., & Höcker, M. (2009). Loss of the coxsackie and adenovirus receptor contributes to gastric cancer progression. BRIT J CANCER, 100(2), 352-359. [2]. http://www.ncbi.nlm.nih.gov/pubmed/19142187?dopt=Citation

Vancouver

Anders M, Vieth M, Röcken C, Ebert M, Pross M, Gretschel S et al. Loss of the coxsackie and adenovirus receptor contributes to gastric cancer progression. BRIT J CANCER. 2009;100(2):352-359. 2.

Bibtex

@article{7b1fea5053ab4a749cc11355529ad17d,
title = "Loss of the coxsackie and adenovirus receptor contributes to gastric cancer progression.",
abstract = "Loss of the coxsackie and adenovirus receptor (CAR) has previously been observed in gastric cancer. The role of CAR in gastric cancer pathobiology, however, is unclear. We therefore analysed CAR in 196 R(0)-resected gastric adenocarcinomas and non-cancerous gastric mucosa samples using immunohistochemistry and immunofluorescence. Coxsackie and adenovirus receptor was found at the surface and foveolar epithelium of all non-neoplastic gastric mucosa samples (n=175), whereas only 56% of gastric cancer specimens showed CAR positivity (P",
author = "Mario Anders and M Vieth and C R{\"o}cken and M Ebert and M Pross and S Gretschel and Schlag, {P M} and B Wiedenmann and W Kemmner and M H{\"o}cker",
year = "2009",
language = "Deutsch",
volume = "100",
pages = "352--359",
journal = "BRIT J CANCER",
issn = "0007-0920",
publisher = "NATURE PUBLISHING GROUP",
number = "2",

}

RIS

TY - JOUR

T1 - Loss of the coxsackie and adenovirus receptor contributes to gastric cancer progression.

AU - Anders, Mario

AU - Vieth, M

AU - Röcken, C

AU - Ebert, M

AU - Pross, M

AU - Gretschel, S

AU - Schlag, P M

AU - Wiedenmann, B

AU - Kemmner, W

AU - Höcker, M

PY - 2009

Y1 - 2009

N2 - Loss of the coxsackie and adenovirus receptor (CAR) has previously been observed in gastric cancer. The role of CAR in gastric cancer pathobiology, however, is unclear. We therefore analysed CAR in 196 R(0)-resected gastric adenocarcinomas and non-cancerous gastric mucosa samples using immunohistochemistry and immunofluorescence. Coxsackie and adenovirus receptor was found at the surface and foveolar epithelium of all non-neoplastic gastric mucosa samples (n=175), whereas only 56% of gastric cancer specimens showed CAR positivity (P

AB - Loss of the coxsackie and adenovirus receptor (CAR) has previously been observed in gastric cancer. The role of CAR in gastric cancer pathobiology, however, is unclear. We therefore analysed CAR in 196 R(0)-resected gastric adenocarcinomas and non-cancerous gastric mucosa samples using immunohistochemistry and immunofluorescence. Coxsackie and adenovirus receptor was found at the surface and foveolar epithelium of all non-neoplastic gastric mucosa samples (n=175), whereas only 56% of gastric cancer specimens showed CAR positivity (P

M3 - SCORING: Zeitschriftenaufsatz

VL - 100

SP - 352

EP - 359

JO - BRIT J CANCER

JF - BRIT J CANCER

SN - 0007-0920

IS - 2

M1 - 2

ER -