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Kallikrein-kinin activation by altered vitreous pH

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Kallikrein-kinin activation by altered vitreous pH : New perspectives for treatment and pathogenesis of diabetic macular edema? : Comment on: Gao BB et al. Extracellular carbonic anhydrase mediates hemorrhagic retinal and cerebral vascular permeability through prekallikrein activation. Nat Med. 2007 Feb;13(2):181-8. / Zeitz, O; Keserü, M.

In: GRAEF ARCH CLIN EXP, Vol. 245, No. 12, 01.12.2007, p. 1745-7.

Research output: SCORING: Contribution to journal › SCORING: Journal article › Research › peer-review

Harvard

Zeitz, O & Keserü, M 2007, 'Kallikrein-kinin activation by altered vitreous pH: New perspectives for treatment and pathogenesis of diabetic macular edema? : Comment on: Gao BB et al. Extracellular carbonic anhydrase mediates hemorrhagic retinal and cerebral vascular permeability through prekallikrein activation. Nat Med. 2007 Feb;13(2):181-8', GRAEF ARCH CLIN EXP, vol. 245, no. 12, pp. 1745-7. https://doi.org/10.1007/s00417-007-0673-7

APA

Zeitz, O., & Keserü, M. (2007). Kallikrein-kinin activation by altered vitreous pH: New perspectives for treatment and pathogenesis of diabetic macular edema? : Comment on: Gao BB et al. Extracellular carbonic anhydrase mediates hemorrhagic retinal and cerebral vascular permeability through prekallikrein activation. Nat Med. 2007 Feb;13(2):181-8. GRAEF ARCH CLIN EXP, 245(12), 1745-7. https://doi.org/10.1007/s00417-007-0673-7

Vancouver

Zeitz O, Keserü M. Kallikrein-kinin activation by altered vitreous pH: New perspectives for treatment and pathogenesis of diabetic macular edema? : Comment on: Gao BB et al. Extracellular carbonic anhydrase mediates hemorrhagic retinal and cerebral vascular permeability through prekallikrein activation. Nat Med. 2007 Feb;13(2):181-8. GRAEF ARCH CLIN EXP. 2007 Dec 1;245(12):1745-7. https://doi.org/10.1007/s00417-007-0673-7

Bibtex

@article{3d70426512544300a1355ce284ab3fd3,
title = "Kallikrein-kinin activation by altered vitreous pH: New perspectives for treatment and pathogenesis of diabetic macular edema? : Comment on: Gao BB et al. Extracellular carbonic anhydrase mediates hemorrhagic retinal and cerebral vascular permeability through prekallikrein activation. Nat Med. 2007 Feb;13(2):181-8",
abstract = "Recently, Gao et al. published an experimental study based on the pathophysiology of diabetic macular edema in Nature Medicine (Nat Med. 2007 Feb;13(2):181-8). They found an increased amount of carbonic anhydrase in the vitreous, which causes a pH shift. This activates the kallikrein-kinin system and leads to increased retinal vascular permeability. In this comment the clinical implications of this article are discussed.",
keywords = "Capillary Permeability, Carbonic Anhydrases, Diabetic Retinopathy, Humans, Hydrogen-Ion Concentration, Kallikrein-Kinin System, Macular Edema, Retinal Vessels, Vitreous Body",
author = "O Zeitz and M Keser{\"u}",
year = "2007",
month = dec,
day = "1",
doi = "10.1007/s00417-007-0673-7",
language = "English",
volume = "245",
pages = "1745--7",
journal = "GRAEF ARCH CLIN EXP",
issn = "0721-832X",
publisher = "Springer",
number = "12",

}

RIS

TY - JOUR

T1 - Kallikrein-kinin activation by altered vitreous pH

T2 - New perspectives for treatment and pathogenesis of diabetic macular edema? : Comment on: Gao BB et al. Extracellular carbonic anhydrase mediates hemorrhagic retinal and cerebral vascular permeability through prekallikrein activation. Nat Med. 2007 Feb;13(2):181-8

AU - Zeitz, O

AU - Keserü, M

PY - 2007/12/1

Y1 - 2007/12/1

N2 - Recently, Gao et al. published an experimental study based on the pathophysiology of diabetic macular edema in Nature Medicine (Nat Med. 2007 Feb;13(2):181-8). They found an increased amount of carbonic anhydrase in the vitreous, which causes a pH shift. This activates the kallikrein-kinin system and leads to increased retinal vascular permeability. In this comment the clinical implications of this article are discussed.

AB - Recently, Gao et al. published an experimental study based on the pathophysiology of diabetic macular edema in Nature Medicine (Nat Med. 2007 Feb;13(2):181-8). They found an increased amount of carbonic anhydrase in the vitreous, which causes a pH shift. This activates the kallikrein-kinin system and leads to increased retinal vascular permeability. In this comment the clinical implications of this article are discussed.

KW - Capillary Permeability

KW - Carbonic Anhydrases

KW - Diabetic Retinopathy

KW - Humans

KW - Hydrogen-Ion Concentration

KW - Kallikrein-Kinin System

KW - Macular Edema

KW - Retinal Vessels

KW - Vitreous Body

U2 - 10.1007/s00417-007-0673-7

DO - 10.1007/s00417-007-0673-7

M3 - SCORING: Journal article

C2 - 17823811

VL - 245

SP - 1745

EP - 1747

JO - GRAEF ARCH CLIN EXP

JF - GRAEF ARCH CLIN EXP

SN - 0721-832X

IS - 12

ER -