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Human obesity reduces the number of hepatic leptin receptor (ob-R) expressing NK cells.

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Human obesity reduces the number of hepatic leptin receptor (ob-R) expressing NK cells. / Lautenbach, Anne; Breitmeier, Dirk; Kuhlmann, Susanne; Nave, Heike.

In: ENDOCR RES, Vol. 36, No. 4, 4, 2011, p. 158-166.

Research output: SCORING: Contribution to journal › SCORING: Journal article › Research › peer-review

Harvard

Lautenbach, A, Breitmeier, D, Kuhlmann, S & Nave, H 2011, 'Human obesity reduces the number of hepatic leptin receptor (ob-R) expressing NK cells.', ENDOCR RES, vol. 36, no. 4, 4, pp. 158-166. <http://www.ncbi.nlm.nih.gov/pubmed/21973235?dopt=Citation>

APA

Lautenbach, A., Breitmeier, D., Kuhlmann, S., & Nave, H. (2011). Human obesity reduces the number of hepatic leptin receptor (ob-R) expressing NK cells. ENDOCR RES, 36(4), 158-166. [4]. http://www.ncbi.nlm.nih.gov/pubmed/21973235?dopt=Citation

Vancouver

Lautenbach A, Breitmeier D, Kuhlmann S, Nave H. Human obesity reduces the number of hepatic leptin receptor (ob-R) expressing NK cells. ENDOCR RES. 2011;36(4):158-166. 4.

Bibtex

@article{a0c2315ac58b43fb94a80d80cf0183c4,
title = "Human obesity reduces the number of hepatic leptin receptor (ob-R) expressing NK cells.",
abstract = "In the industrialized world, obesity is an increasing socioeconomic health problem. Obese subjects have a higher risk of developing several types of cancer. NK cells are an integral component of the innate immune system, able to destruct tumor cells. The adipokine leptin plays a crucial role in the development of obesity and its related diseases. Peripheral leptin signaling is modulated by the liver.",
keywords = "Adult, Humans, Aged, Middle Aged, Adolescent, Young Adult, Immunohistochemistry, Cell Count, Liver/metabolism/pathology, Killer Cells, Natural/*metabolism/pathology, Obesity/*metabolism/*pathology, Receptors, Leptin/*biosynthesis, Adult, Humans, Aged, Middle Aged, Adolescent, Young Adult, Immunohistochemistry, Cell Count, Liver/metabolism/pathology, Killer Cells, Natural/*metabolism/pathology, Obesity/*metabolism/*pathology, Receptors, Leptin/*biosynthesis",
author = "Anne Lautenbach and Dirk Breitmeier and Susanne Kuhlmann and Heike Nave",
year = "2011",
language = "English",
volume = "36",
pages = "158--166",
journal = "ENDOCR RES",
issn = "0743-5800",
publisher = "informa healthcare",
number = "4",

}

RIS

TY - JOUR

T1 - Human obesity reduces the number of hepatic leptin receptor (ob-R) expressing NK cells.

AU - Lautenbach, Anne

AU - Breitmeier, Dirk

AU - Kuhlmann, Susanne

AU - Nave, Heike

PY - 2011

Y1 - 2011

N2 - In the industrialized world, obesity is an increasing socioeconomic health problem. Obese subjects have a higher risk of developing several types of cancer. NK cells are an integral component of the innate immune system, able to destruct tumor cells. The adipokine leptin plays a crucial role in the development of obesity and its related diseases. Peripheral leptin signaling is modulated by the liver.

AB - In the industrialized world, obesity is an increasing socioeconomic health problem. Obese subjects have a higher risk of developing several types of cancer. NK cells are an integral component of the innate immune system, able to destruct tumor cells. The adipokine leptin plays a crucial role in the development of obesity and its related diseases. Peripheral leptin signaling is modulated by the liver.

KW - Adult

KW - Humans

KW - Aged

KW - Middle Aged

KW - Adolescent

KW - Young Adult

KW - Immunohistochemistry

KW - Cell Count

KW - Liver/metabolism/pathology

KW - Killer Cells, Natural/metabolism/pathology

KW - Obesity/metabolism/pathology

KW - Receptors, Leptin/biosynthesis

KW - Adult

KW - Humans

KW - Aged

KW - Middle Aged

KW - Adolescent

KW - Young Adult

KW - Immunohistochemistry

KW - Cell Count

KW - Liver/metabolism/pathology

KW - Killer Cells, Natural/metabolism/pathology

KW - Obesity/metabolism/pathology

KW - Receptors, Leptin/biosynthesis

M3 - SCORING: Journal article

VL - 36

SP - 158

EP - 166

JO - ENDOCR RES

JF - ENDOCR RES

SN - 0743-5800

IS - 4

M1 - 4

ER -