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Effect of kidney disease on glucose handling (including genetic defects)

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Effect of kidney disease on glucose handling (including genetic defects). / Calado, Joaquim; Santer, René; Rueff, José.

In: Kidney international. Supplement, No. 120, 03.2011, p. S7-13.

Research output: SCORING: Contribution to journal › SCORING: Review article › Research

Harvard

Calado, J, Santer, R & Rueff, J 2011, 'Effect of kidney disease on glucose handling (including genetic defects)', Kidney international. Supplement, no. 120, pp. S7-13. https://doi.org/10.1038/ki.2010.510

APA

Calado, J., Santer, R., & Rueff, J. (2011). Effect of kidney disease on glucose handling (including genetic defects). Kidney international. Supplement, (120), S7-13. https://doi.org/10.1038/ki.2010.510

Vancouver

Calado J, Santer R, Rueff J. Effect of kidney disease on glucose handling (including genetic defects). Kidney international. Supplement. 2011 Mar;(120):S7-13. https://doi.org/10.1038/ki.2010.510

Bibtex

@article{cabb871b94af49f2ac0732c3269ebfd6,
title = "Effect of kidney disease on glucose handling (including genetic defects)",
abstract = "Reabsorption of glucose in the proximal renal tubule involves the Na(+)-coupled glucose cotransporter (SGLT) and the facilitative glucose transport (GLUT) multigene glucose transport families. Mutations in SLC5A2, the SGLT2 coding gene, are responsible for familial renal glucosuria (FRG), a genetic disorder characterized by glucosuria in the absence of both hyperglycemia and generalized proximal tubular dysfunction. In this paper we focus on FRG and describe other inherited and acquired clinical conditions associated with glucosuria. In addition, a brief review on the regulation of renal glucose transport in diabetes is provided.",
keywords = "Animals, Genetic Association Studies, Glycosuria/genetics, Humans, Kidney Diseases/genetics, Malabsorption Syndromes, Mice, Monosaccharide Transport Proteins/genetics",
author = "Joaquim Calado and Ren{\'e} Santer and Jos{\'e} Rueff",
year = "2011",
month = mar,
doi = "10.1038/ki.2010.510",
language = "English",
pages = "S7--13",
number = "120",

}

RIS

TY - JOUR

T1 - Effect of kidney disease on glucose handling (including genetic defects)

AU - Calado, Joaquim

AU - Santer, René

AU - Rueff, José

PY - 2011/3

Y1 - 2011/3

N2 - Reabsorption of glucose in the proximal renal tubule involves the Na(+)-coupled glucose cotransporter (SGLT) and the facilitative glucose transport (GLUT) multigene glucose transport families. Mutations in SLC5A2, the SGLT2 coding gene, are responsible for familial renal glucosuria (FRG), a genetic disorder characterized by glucosuria in the absence of both hyperglycemia and generalized proximal tubular dysfunction. In this paper we focus on FRG and describe other inherited and acquired clinical conditions associated with glucosuria. In addition, a brief review on the regulation of renal glucose transport in diabetes is provided.

AB - Reabsorption of glucose in the proximal renal tubule involves the Na(+)-coupled glucose cotransporter (SGLT) and the facilitative glucose transport (GLUT) multigene glucose transport families. Mutations in SLC5A2, the SGLT2 coding gene, are responsible for familial renal glucosuria (FRG), a genetic disorder characterized by glucosuria in the absence of both hyperglycemia and generalized proximal tubular dysfunction. In this paper we focus on FRG and describe other inherited and acquired clinical conditions associated with glucosuria. In addition, a brief review on the regulation of renal glucose transport in diabetes is provided.

KW - Animals

KW - Genetic Association Studies

KW - Glycosuria/genetics

KW - Humans

KW - Kidney Diseases/genetics

KW - Malabsorption Syndromes

KW - Mice

KW - Monosaccharide Transport Proteins/genetics

U2 - 10.1038/ki.2010.510

DO - 10.1038/ki.2010.510

M3 - SCORING: Review article

C2 - 21358700

SP - S7-13

IS - 120

ER -