Research ExplorerPublications[Cyclooxygenase inhibitors: adverse drug reactions are unavo...

[Cyclooxygenase inhibitors: adverse drug reactions are unavoidable, but only a few are (acutely) dangerous]

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[Cyclooxygenase inhibitors: adverse drug reactions are unavoidable, but only a few are (acutely) dangerous]. / Brune, K; Renner, B; Maas, Renke.

In: DEUT MED WOCHENSCHR, Vol. 134, No. 36, 36, 2009, p. 1771-1773.

Research output: SCORING: Contribution to journal › SCORING: Journal article › Research › peer-review

Harvard

Brune, K, Renner, B & Maas, R 2009, '[Cyclooxygenase inhibitors: adverse drug reactions are unavoidable, but only a few are (acutely) dangerous]', DEUT MED WOCHENSCHR, vol. 134, no. 36, 36, pp. 1771-1773. <http://www.ncbi.nlm.nih.gov/pubmed/19718601?dopt=Citation>

APA

Brune, K., Renner, B., & Maas, R. (2009). [Cyclooxygenase inhibitors: adverse drug reactions are unavoidable, but only a few are (acutely) dangerous]. DEUT MED WOCHENSCHR, 134(36), 1771-1773. [36]. http://www.ncbi.nlm.nih.gov/pubmed/19718601?dopt=Citation

Vancouver

Brune K, Renner B, Maas R. [Cyclooxygenase inhibitors: adverse drug reactions are unavoidable, but only a few are (acutely) dangerous]. DEUT MED WOCHENSCHR. 2009;134(36):1771-1773. 36.

Bibtex

@article{39482795155347e3bc7f9cd0012202e4,
title = "[Cyclooxygenase inhibitors: adverse drug reactions are unavoidable, but only a few are (acutely) dangerous]",
abstract = "Non-steroidal, anti-inflammatory drugs are the most widely used remedies worldwide. Drug interactions are frequent, life threatening ones are rare: Bleeds on the basis of inhibition of platelet aggregation and loss of organ protection (GI-tract), thrombotic events due to loss of vasoprotection and increased propensity for blood coagulation (stroke), acute kidney failure on the basis of loss of kidney protection by prostaglandins together with stress to the organ and/or drugs interfering with the renal blood flow. Acute liver toxicity is typical for acetaminophen. Whether the risk is increased by other hepatotoxic drugs, as diclofenac, is uncertain at present. All risks can be reduced considerably by avoiding problematic drug interactions.",
keywords = "Humans, Cyclooxygenase Inhibitors adverse effects, Hemorrhage chemically induced, Intestinal Perforation chemically induced, Kidney drug effects, Kidney Failure chemically induced, Liver drug effects, Liver Failure chemically induced, Platelet Aggregation drug effects, Thromboembolism chemically induced, Humans, Cyclooxygenase Inhibitors adverse effects, Hemorrhage chemically induced, Intestinal Perforation chemically induced, Kidney drug effects, Kidney Failure chemically induced, Liver drug effects, Liver Failure chemically induced, Platelet Aggregation drug effects, Thromboembolism chemically induced",
author = "K Brune and B Renner and Renke Maas",
year = "2009",
language = "Deutsch",
volume = "134",
pages = "1771--1773",
journal = "DEUT MED WOCHENSCHR",
issn = "0012-0472",
publisher = "Georg Thieme Verlag KG",
number = "36",

}

RIS

TY - JOUR

T1 - [Cyclooxygenase inhibitors: adverse drug reactions are unavoidable, but only a few are (acutely) dangerous]

AU - Brune, K

AU - Renner, B

AU - Maas, Renke

PY - 2009

Y1 - 2009

N2 - Non-steroidal, anti-inflammatory drugs are the most widely used remedies worldwide. Drug interactions are frequent, life threatening ones are rare: Bleeds on the basis of inhibition of platelet aggregation and loss of organ protection (GI-tract), thrombotic events due to loss of vasoprotection and increased propensity for blood coagulation (stroke), acute kidney failure on the basis of loss of kidney protection by prostaglandins together with stress to the organ and/or drugs interfering with the renal blood flow. Acute liver toxicity is typical for acetaminophen. Whether the risk is increased by other hepatotoxic drugs, as diclofenac, is uncertain at present. All risks can be reduced considerably by avoiding problematic drug interactions.

AB - Non-steroidal, anti-inflammatory drugs are the most widely used remedies worldwide. Drug interactions are frequent, life threatening ones are rare: Bleeds on the basis of inhibition of platelet aggregation and loss of organ protection (GI-tract), thrombotic events due to loss of vasoprotection and increased propensity for blood coagulation (stroke), acute kidney failure on the basis of loss of kidney protection by prostaglandins together with stress to the organ and/or drugs interfering with the renal blood flow. Acute liver toxicity is typical for acetaminophen. Whether the risk is increased by other hepatotoxic drugs, as diclofenac, is uncertain at present. All risks can be reduced considerably by avoiding problematic drug interactions.

KW - Humans

KW - Cyclooxygenase Inhibitors adverse effects

KW - Hemorrhage chemically induced

KW - Intestinal Perforation chemically induced

KW - Kidney drug effects

KW - Kidney Failure chemically induced

KW - Liver drug effects

KW - Liver Failure chemically induced

KW - Platelet Aggregation drug effects

KW - Thromboembolism chemically induced

KW - Humans

KW - Cyclooxygenase Inhibitors adverse effects

KW - Hemorrhage chemically induced

KW - Intestinal Perforation chemically induced

KW - Kidney drug effects

KW - Kidney Failure chemically induced

KW - Liver drug effects

KW - Liver Failure chemically induced

KW - Platelet Aggregation drug effects

KW - Thromboembolism chemically induced

M3 - SCORING: Zeitschriftenaufsatz

VL - 134

SP - 1771

EP - 1773

JO - DEUT MED WOCHENSCHR

JF - DEUT MED WOCHENSCHR

SN - 0012-0472

IS - 36

M1 - 36

ER -