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Yersinia virulence factor YopM induces sustained RSK activation by interfering with dephosphorylation.

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Yersinia virulence factor YopM induces sustained RSK activation by interfering with dephosphorylation. / Hentschke, Moritz; Berneking, Laura; Campos, Cristina Belmar; Buck, Friedrich; Ruckdeschel, Klaus; Aepfelbacher, Martin.

in: PLOS ONE, Jahrgang 5, Nr. 10, 10, 2010.

Publikationen: SCORING: Beitrag in Fachzeitschrift/Zeitung › SCORING: Zeitschriftenaufsatz › Forschung › Begutachtung

Harvard

Hentschke, M, Berneking, L, Campos, CB, Buck, F, Ruckdeschel, K & Aepfelbacher, M 2010, 'Yersinia virulence factor YopM induces sustained RSK activation by interfering with dephosphorylation.', PLOS ONE, Jg. 5, Nr. 10, 10. https://doi.org/10.1371/journal.pone.0013165

APA

Hentschke, M., Berneking, L., Campos, C. B., Buck, F., Ruckdeschel, K., & Aepfelbacher, M. (2010). Yersinia virulence factor YopM induces sustained RSK activation by interfering with dephosphorylation. PLOS ONE, 5(10), [10]. https://doi.org/10.1371/journal.pone.0013165

Vancouver

Hentschke M, Berneking L, Campos CB, Buck F, Ruckdeschel K, Aepfelbacher M. Yersinia virulence factor YopM induces sustained RSK activation by interfering with dephosphorylation. PLOS ONE. 2010;5(10). 10. https://doi.org/10.1371/journal.pone.0013165

Bibtex

@article{336e759b6c5c449282a03075448cceb8,
title = "Yersinia virulence factor YopM induces sustained RSK activation by interfering with dephosphorylation.",
abstract = "Pathogenic yersiniae inject several effector proteins (Yops) into host cells, which subverts immune functions and enables the bacteria to survive within the host organism. YopM, whose deletion in enteropathogenic yersiniae results in a dramatic loss of virulence, has previously been shown to form a complex with and activate the multifunctional kinases PKN2 and RSK1 in transfected cells.",
author = "Moritz Hentschke and Laura Berneking and Campos, {Cristina Belmar} and Friedrich Buck and Klaus Ruckdeschel and Martin Aepfelbacher",
year = "2010",
doi = "10.1371/journal.pone.0013165",
language = "Deutsch",
volume = "5",
journal = "PLOS ONE",
issn = "1932-6203",
publisher = "Public Library of Science",
number = "10",

}

RIS

TY - JOUR

T1 - Yersinia virulence factor YopM induces sustained RSK activation by interfering with dephosphorylation.

AU - Hentschke, Moritz

AU - Berneking, Laura

AU - Campos, Cristina Belmar

AU - Buck, Friedrich

AU - Ruckdeschel, Klaus

AU - Aepfelbacher, Martin

PY - 2010

Y1 - 2010

N2 - Pathogenic yersiniae inject several effector proteins (Yops) into host cells, which subverts immune functions and enables the bacteria to survive within the host organism. YopM, whose deletion in enteropathogenic yersiniae results in a dramatic loss of virulence, has previously been shown to form a complex with and activate the multifunctional kinases PKN2 and RSK1 in transfected cells.

AB - Pathogenic yersiniae inject several effector proteins (Yops) into host cells, which subverts immune functions and enables the bacteria to survive within the host organism. YopM, whose deletion in enteropathogenic yersiniae results in a dramatic loss of virulence, has previously been shown to form a complex with and activate the multifunctional kinases PKN2 and RSK1 in transfected cells.

U2 - 10.1371/journal.pone.0013165

DO - 10.1371/journal.pone.0013165

M3 - SCORING: Zeitschriftenaufsatz

VL - 5

JO - PLOS ONE

JF - PLOS ONE

SN - 1932-6203

IS - 10

M1 - 10

ER -