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Nogo CAA 3'UTR Insertion polymorphism is not associated with Schizophrenia nor with bipolar disorder.

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Nogo CAA 3'UTR Insertion polymorphism is not associated with Schizophrenia nor with bipolar disorder. / Gregório, Sheila P; Mury, Fábio B; Ojopi, Elida B; Sallet, Paulo C; Moreno, Doris H; Yacubian, Juliana; Tavares, Hildeberto; Santos, Fabrício R; Gattaz, Wagner F; Dias-Neto, Emmanuel.

in: SCHIZOPHR RES, Jahrgang 75, Nr. 1, 1, 2005, S. 5-9.

Publikationen: SCORING: Beitrag in Fachzeitschrift/Zeitung › SCORING: Zeitschriftenaufsatz › Forschung › Begutachtung

Harvard

Gregório, SP, Mury, FB, Ojopi, EB, Sallet, PC, Moreno, DH, Yacubian, J, Tavares, H, Santos, FR, Gattaz, WF & Dias-Neto, E 2005, 'Nogo CAA 3'UTR Insertion polymorphism is not associated with Schizophrenia nor with bipolar disorder.', SCHIZOPHR RES, Jg. 75, Nr. 1, 1, S. 5-9. <http://www.ncbi.nlm.nih.gov/pubmed/15820318?dopt=Citation>

APA

Gregório, S. P., Mury, F. B., Ojopi, E. B., Sallet, P. C., Moreno, D. H., Yacubian, J., Tavares, H., Santos, F. R., Gattaz, W. F., & Dias-Neto, E. (2005). Nogo CAA 3'UTR Insertion polymorphism is not associated with Schizophrenia nor with bipolar disorder. SCHIZOPHR RES, 75(1), 5-9. [1]. http://www.ncbi.nlm.nih.gov/pubmed/15820318?dopt=Citation

Vancouver

Gregório SP, Mury FB, Ojopi EB, Sallet PC, Moreno DH, Yacubian J et al. Nogo CAA 3'UTR Insertion polymorphism is not associated with Schizophrenia nor with bipolar disorder. SCHIZOPHR RES. 2005;75(1):5-9. 1.

Bibtex

@article{86bc97e5bb73496e9deaa68cd0900137,
title = "Nogo CAA 3'UTR Insertion polymorphism is not associated with Schizophrenia nor with bipolar disorder.",
abstract = "The Nogo gene maps to 2p14-p13, a region consistently associated with schizophrenia and bipolar disorder. The association of a polymorphism in Nogo was previously investigated by two groups, with divergent results. In this report, using an alternative approach, we evaluated this same polymorphism in 725 individuals, including patients with schizophrenia, bipolar disorder, normal controls and non-human primate samples. Our results indicate that the polymorphism is not associated with any of these diseases, but has a remarkably biased distribution in ethnic groups. Genotyping of primate samples, suggest that this polymorphism is a recent event in human speciation.",
author = "Greg{\'o}rio, {Sheila P} and Mury, {F{\'a}bio B} and Ojopi, {Elida B} and Sallet, {Paulo C} and Moreno, {Doris H} and Juliana Yacubian and Hildeberto Tavares and Santos, {Fabr{\'i}cio R} and Gattaz, {Wagner F} and Emmanuel Dias-Neto",
year = "2005",
language = "Deutsch",
volume = "75",
pages = "5--9",
journal = "SCHIZOPHR RES",
issn = "0920-9964",
publisher = "Elsevier",
number = "1",

}

RIS

TY - JOUR

T1 - Nogo CAA 3'UTR Insertion polymorphism is not associated with Schizophrenia nor with bipolar disorder.

AU - Gregório, Sheila P

AU - Mury, Fábio B

AU - Ojopi, Elida B

AU - Sallet, Paulo C

AU - Moreno, Doris H

AU - Yacubian, Juliana

AU - Tavares, Hildeberto

AU - Santos, Fabrício R

AU - Gattaz, Wagner F

AU - Dias-Neto, Emmanuel

PY - 2005

Y1 - 2005

N2 - The Nogo gene maps to 2p14-p13, a region consistently associated with schizophrenia and bipolar disorder. The association of a polymorphism in Nogo was previously investigated by two groups, with divergent results. In this report, using an alternative approach, we evaluated this same polymorphism in 725 individuals, including patients with schizophrenia, bipolar disorder, normal controls and non-human primate samples. Our results indicate that the polymorphism is not associated with any of these diseases, but has a remarkably biased distribution in ethnic groups. Genotyping of primate samples, suggest that this polymorphism is a recent event in human speciation.

AB - The Nogo gene maps to 2p14-p13, a region consistently associated with schizophrenia and bipolar disorder. The association of a polymorphism in Nogo was previously investigated by two groups, with divergent results. In this report, using an alternative approach, we evaluated this same polymorphism in 725 individuals, including patients with schizophrenia, bipolar disorder, normal controls and non-human primate samples. Our results indicate that the polymorphism is not associated with any of these diseases, but has a remarkably biased distribution in ethnic groups. Genotyping of primate samples, suggest that this polymorphism is a recent event in human speciation.

M3 - SCORING: Zeitschriftenaufsatz

VL - 75

SP - 5

EP - 9

JO - SCHIZOPHR RES

JF - SCHIZOPHR RES

SN - 0920-9964

IS - 1

M1 - 1

ER -