PortalPublikationenIn tumor cells regulation of DNA double strand break repair ...

In tumor cells regulation of DNA double strand break repair through EGF receptor involves both NHEJ and HR and is independent of p53 and K-Ras status.

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In tumor cells regulation of DNA double strand break repair through EGF receptor involves both NHEJ and HR and is independent of p53 and K-Ras status. / Myllynen, Laura; Rieckmann, Thorsten; Dahm-Daphi, Jochen; Kasten-Pisula, Ulla; Petersen, Cordula; Dikomey, Ekkehard; Kriegs, Malte.

in: RADIOTHER ONCOL, Jahrgang 101, Nr. 1, 1, 2011, S. 147-151.

Publikationen: SCORING: Beitrag in Fachzeitschrift/Zeitung › SCORING: Zeitschriftenaufsatz › Forschung › Begutachtung

Harvard

Myllynen, L, Rieckmann, T, Dahm-Daphi, J, Kasten-Pisula, U, Petersen, C, Dikomey, E & Kriegs, M 2011, 'In tumor cells regulation of DNA double strand break repair through EGF receptor involves both NHEJ and HR and is independent of p53 and K-Ras status.', RADIOTHER ONCOL, Jg. 101, Nr. 1, 1, S. 147-151. <http://www.ncbi.nlm.nih.gov/pubmed/21665306?dopt=Citation>

APA

Myllynen, L., Rieckmann, T., Dahm-Daphi, J., Kasten-Pisula, U., Petersen, C., Dikomey, E., & Kriegs, M. (2011). In tumor cells regulation of DNA double strand break repair through EGF receptor involves both NHEJ and HR and is independent of p53 and K-Ras status. RADIOTHER ONCOL, 101(1), 147-151. [1]. http://www.ncbi.nlm.nih.gov/pubmed/21665306?dopt=Citation

Vancouver

Myllynen L, Rieckmann T, Dahm-Daphi J, Kasten-Pisula U, Petersen C, Dikomey E et al. In tumor cells regulation of DNA double strand break repair through EGF receptor involves both NHEJ and HR and is independent of p53 and K-Ras status. RADIOTHER ONCOL. 2011;101(1):147-151. 1.

Bibtex

@article{4d833282aa154a74b5a2e20106f33c52,
title = "In tumor cells regulation of DNA double strand break repair through EGF receptor involves both NHEJ and HR and is independent of p53 and K-Ras status.",
abstract = "The purpose of this study was to examine whether the epidermal growth factor receptor (EGFR) may be used as a general target to modulate DNA double strand break (DSB) repair in tumor cells.",
author = "Laura Myllynen and Thorsten Rieckmann and Jochen Dahm-Daphi and Ulla Kasten-Pisula and Cordula Petersen and Ekkehard Dikomey and Malte Kriegs",
year = "2011",
language = "English",
volume = "101",
pages = "147--151",
journal = "RADIOTHER ONCOL",
issn = "0167-8140",
publisher = "Elsevier Ireland Ltd",
number = "1",

}

RIS

TY - JOUR

T1 - In tumor cells regulation of DNA double strand break repair through EGF receptor involves both NHEJ and HR and is independent of p53 and K-Ras status.

AU - Myllynen, Laura

AU - Rieckmann, Thorsten

AU - Dahm-Daphi, Jochen

AU - Kasten-Pisula, Ulla

AU - Petersen, Cordula

AU - Dikomey, Ekkehard

AU - Kriegs, Malte

PY - 2011

Y1 - 2011

N2 - The purpose of this study was to examine whether the epidermal growth factor receptor (EGFR) may be used as a general target to modulate DNA double strand break (DSB) repair in tumor cells.

AB - The purpose of this study was to examine whether the epidermal growth factor receptor (EGFR) may be used as a general target to modulate DNA double strand break (DSB) repair in tumor cells.

M3 - SCORING: Journal article

VL - 101

SP - 147

EP - 151

JO - RADIOTHER ONCOL

JF - RADIOTHER ONCOL

SN - 0167-8140

IS - 1

M1 - 1

ER -