PortalPublikationenEffector functions of pathogenic Yersinia species.

Effector functions of pathogenic Yersinia species.

Standard

Effector functions of pathogenic Yersinia species. / Aepfelbacher, Martin; Trasak, Claudia; Ruckdeschel, Klaus.

in: THROMB HAEMOSTASIS, Jahrgang 98, Nr. 3, 3, 2007, S. 521-529.

Publikationen: SCORING: Beitrag in Fachzeitschrift/Zeitung › SCORING: Zeitschriftenaufsatz › Forschung › Begutachtung

Harvard

Aepfelbacher, M, Trasak, C & Ruckdeschel, K 2007, 'Effector functions of pathogenic Yersinia species.', THROMB HAEMOSTASIS, Jg. 98, Nr. 3, 3, S. 521-529. <http://www.ncbi.nlm.nih.gov/pubmed/17849040?dopt=Citation>

APA

Aepfelbacher, M., Trasak, C., & Ruckdeschel, K. (2007). Effector functions of pathogenic Yersinia species. THROMB HAEMOSTASIS, 98(3), 521-529. [3]. http://www.ncbi.nlm.nih.gov/pubmed/17849040?dopt=Citation

Vancouver

Aepfelbacher M, Trasak C, Ruckdeschel K. Effector functions of pathogenic Yersinia species. THROMB HAEMOSTASIS. 2007;98(3):521-529. 3.

Bibtex

@article{5ec4f395b9da471cb401e5b2004aa515,
title = "Effector functions of pathogenic Yersinia species.",
abstract = "Pathogenic species of the genus Yersinia suppress and reorient the immune system to infect lymphatic tissues, inner organs and at times also the vasculature. For this purpose yersiniae employ a type III secretion system to translocate effector proteins (Yersinia outer proteins; Yops) into immune cells. Yops often exert unique biochemical activities for modulating the activity of Rho GTP-binding proteins, focal adhesion proteins, inflammatory pathways and cell survival/apoptosis. In this review we will put emphasis on the biochemistry, cell- and infection biology of Yersinia effector Yops.",
author = "Martin Aepfelbacher and Claudia Trasak and Klaus Ruckdeschel",
year = "2007",
language = "Deutsch",
volume = "98",
pages = "521--529",
journal = "THROMB HAEMOSTASIS",
issn = "0340-6245",
publisher = "Schattauer",
number = "3",

}

RIS

TY - JOUR

T1 - Effector functions of pathogenic Yersinia species.

AU - Aepfelbacher, Martin

AU - Trasak, Claudia

AU - Ruckdeschel, Klaus

PY - 2007

Y1 - 2007

N2 - Pathogenic species of the genus Yersinia suppress and reorient the immune system to infect lymphatic tissues, inner organs and at times also the vasculature. For this purpose yersiniae employ a type III secretion system to translocate effector proteins (Yersinia outer proteins; Yops) into immune cells. Yops often exert unique biochemical activities for modulating the activity of Rho GTP-binding proteins, focal adhesion proteins, inflammatory pathways and cell survival/apoptosis. In this review we will put emphasis on the biochemistry, cell- and infection biology of Yersinia effector Yops.

AB - Pathogenic species of the genus Yersinia suppress and reorient the immune system to infect lymphatic tissues, inner organs and at times also the vasculature. For this purpose yersiniae employ a type III secretion system to translocate effector proteins (Yersinia outer proteins; Yops) into immune cells. Yops often exert unique biochemical activities for modulating the activity of Rho GTP-binding proteins, focal adhesion proteins, inflammatory pathways and cell survival/apoptosis. In this review we will put emphasis on the biochemistry, cell- and infection biology of Yersinia effector Yops.

M3 - SCORING: Zeitschriftenaufsatz

VL - 98

SP - 521

EP - 529

JO - THROMB HAEMOSTASIS

JF - THROMB HAEMOSTASIS

SN - 0340-6245

IS - 3

M1 - 3

ER -