Dying-back oligodendrogliopathy

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Dying-back oligodendrogliopathy : a late sequel of myelin-associated glycoprotein deficiency. / Lassmann, H; Bartsch, U; Montag, D; Schachner, M.

in: GLIA, Jahrgang 19, Nr. 2, 02.1997, S. 104-10.

Publikationen: SCORING: Beitrag in Fachzeitschrift/ZeitungSCORING: ZeitschriftenaufsatzForschungBegutachtung

Harvard

Lassmann, H, Bartsch, U, Montag, D & Schachner, M 1997, 'Dying-back oligodendrogliopathy: a late sequel of myelin-associated glycoprotein deficiency', GLIA, Jg. 19, Nr. 2, S. 104-10.

APA

Vancouver

Bibtex

@article{258a8ffa845541e09e2310821f724423,
title = "Dying-back oligodendrogliopathy: a late sequel of myelin-associated glycoprotein deficiency",
abstract = "Ultrastructural analysis of myelin from 8-month-old mice deficient in the myelin-associated glycoprotein revealed pronounced and characteristic alterations of the periaxonal oligodendrocyte processes, consisting of intracytoplasmic deposition of vesicular material, multivesicular bodies, mitochondria, and lipofuscin granules, as well as granular or paracrystalline inclusions. These alterations are similar to those described before as {"}dying-back oligodendrogliopathy{"} in diseases of toxic or immune-mediated demyelination including multiple sclerosis.",
keywords = "Animals, Cell Death, Mice, Mice, Mutant Strains, Microscopy, Electron, Mutation, Myelin-Associated Glycoprotein, Nerve Fibers, Myelinated, Nervous System Diseases, Oligodendroglia, Journal Article, Research Support, Non-U.S. Gov't",
author = "H Lassmann and U Bartsch and D Montag and M Schachner",
year = "1997",
month = feb,
language = "English",
volume = "19",
pages = "104--10",
journal = "GLIA",
issn = "0894-1491",
publisher = "John Wiley and Sons Inc.",
number = "2",

}

RIS

TY - JOUR

T1 - Dying-back oligodendrogliopathy

T2 - a late sequel of myelin-associated glycoprotein deficiency

AU - Lassmann, H

AU - Bartsch, U

AU - Montag, D

AU - Schachner, M

PY - 1997/2

Y1 - 1997/2

N2 - Ultrastructural analysis of myelin from 8-month-old mice deficient in the myelin-associated glycoprotein revealed pronounced and characteristic alterations of the periaxonal oligodendrocyte processes, consisting of intracytoplasmic deposition of vesicular material, multivesicular bodies, mitochondria, and lipofuscin granules, as well as granular or paracrystalline inclusions. These alterations are similar to those described before as "dying-back oligodendrogliopathy" in diseases of toxic or immune-mediated demyelination including multiple sclerosis.

AB - Ultrastructural analysis of myelin from 8-month-old mice deficient in the myelin-associated glycoprotein revealed pronounced and characteristic alterations of the periaxonal oligodendrocyte processes, consisting of intracytoplasmic deposition of vesicular material, multivesicular bodies, mitochondria, and lipofuscin granules, as well as granular or paracrystalline inclusions. These alterations are similar to those described before as "dying-back oligodendrogliopathy" in diseases of toxic or immune-mediated demyelination including multiple sclerosis.

KW - Animals

KW - Cell Death

KW - Mice

KW - Mice, Mutant Strains

KW - Microscopy, Electron

KW - Mutation

KW - Myelin-Associated Glycoprotein

KW - Nerve Fibers, Myelinated

KW - Nervous System Diseases

KW - Oligodendroglia

KW - Journal Article

KW - Research Support, Non-U.S. Gov't

M3 - SCORING: Journal article

C2 - 9034827

VL - 19

SP - 104

EP - 110

JO - GLIA

JF - GLIA

SN - 0894-1491

IS - 2

ER -