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Decrease in adhesion molecules on polymorphonuclear leukocytes of patients with fibromyalgia.

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Decrease in adhesion molecules on polymorphonuclear leukocytes of patients with fibromyalgia. / Kaufmann, Ines; Schelling, Gustav; Eisner, Christoph; Richter, Hans Peter; Beyer, Antje; Krauseneck, Till; Chouker, Alexander; Thiel, Manfred.

in: RHEUMATOL INT, Jahrgang 29, Nr. 9, 9, 2009, S. 1109-1111.

Publikationen: SCORING: Beitrag in Fachzeitschrift/Zeitung › SCORING: Zeitschriftenaufsatz › Forschung › Begutachtung

Harvard

Kaufmann, I, Schelling, G, Eisner, C, Richter, HP, Beyer, A, Krauseneck, T, Chouker, A & Thiel, M 2009, 'Decrease in adhesion molecules on polymorphonuclear leukocytes of patients with fibromyalgia.', RHEUMATOL INT, Jg. 29, Nr. 9, 9, S. 1109-1111. <http://www.ncbi.nlm.nih.gov/pubmed/19052753?dopt=Citation>

APA

Kaufmann, I., Schelling, G., Eisner, C., Richter, H. P., Beyer, A., Krauseneck, T., Chouker, A., & Thiel, M. (2009). Decrease in adhesion molecules on polymorphonuclear leukocytes of patients with fibromyalgia. RHEUMATOL INT, 29(9), 1109-1111. [9]. http://www.ncbi.nlm.nih.gov/pubmed/19052753?dopt=Citation

Vancouver

Kaufmann I, Schelling G, Eisner C, Richter HP, Beyer A, Krauseneck T et al. Decrease in adhesion molecules on polymorphonuclear leukocytes of patients with fibromyalgia. RHEUMATOL INT. 2009;29(9):1109-1111. 9.

Bibtex

@article{f02464f30cab4a90b6d3ee306b9fe70d,
title = "Decrease in adhesion molecules on polymorphonuclear leukocytes of patients with fibromyalgia.",
abstract = "Fibromyalgia (FM) is a chronic widespread pain condition in highly stressed humans. Because stress is known to modulate adhesion molecule expression, we determined L: -selectin (CD62L) and beta(2)-integrin (CD11b/CD18) expression on the surface of polymorphonuclear leukocytes in 22 patients with FM. As compared to age and sex-matched healthy controls, FM patients showed a significantly decreased expression of CD62L (p <0.01) and CD11b/CD18 (p <0.05) on polymorphonuclear leukocytes. These changes might lower the rate of polymorphonuclear leukocyte migration to sites of inflammation and thereby compromise defense against infections and pain control.",
author = "Ines Kaufmann and Gustav Schelling and Christoph Eisner and Richter, {Hans Peter} and Antje Beyer and Till Krauseneck and Alexander Chouker and Manfred Thiel",
year = "2009",
language = "Deutsch",
volume = "29",
pages = "1109--1111",
journal = "RHEUMATOL INT",
issn = "0172-8172",
publisher = "Springer",
number = "9",

}

RIS

TY - JOUR

T1 - Decrease in adhesion molecules on polymorphonuclear leukocytes of patients with fibromyalgia.

AU - Kaufmann, Ines

AU - Schelling, Gustav

AU - Eisner, Christoph

AU - Richter, Hans Peter

AU - Beyer, Antje

AU - Krauseneck, Till

AU - Chouker, Alexander

AU - Thiel, Manfred

PY - 2009

Y1 - 2009

N2 - Fibromyalgia (FM) is a chronic widespread pain condition in highly stressed humans. Because stress is known to modulate adhesion molecule expression, we determined L: -selectin (CD62L) and beta(2)-integrin (CD11b/CD18) expression on the surface of polymorphonuclear leukocytes in 22 patients with FM. As compared to age and sex-matched healthy controls, FM patients showed a significantly decreased expression of CD62L (p <0.01) and CD11b/CD18 (p <0.05) on polymorphonuclear leukocytes. These changes might lower the rate of polymorphonuclear leukocyte migration to sites of inflammation and thereby compromise defense against infections and pain control.

AB - Fibromyalgia (FM) is a chronic widespread pain condition in highly stressed humans. Because stress is known to modulate adhesion molecule expression, we determined L: -selectin (CD62L) and beta(2)-integrin (CD11b/CD18) expression on the surface of polymorphonuclear leukocytes in 22 patients with FM. As compared to age and sex-matched healthy controls, FM patients showed a significantly decreased expression of CD62L (p <0.01) and CD11b/CD18 (p <0.05) on polymorphonuclear leukocytes. These changes might lower the rate of polymorphonuclear leukocyte migration to sites of inflammation and thereby compromise defense against infections and pain control.

M3 - SCORING: Zeitschriftenaufsatz

VL - 29

SP - 1109

EP - 1111

JO - RHEUMATOL INT

JF - RHEUMATOL INT

SN - 0172-8172

IS - 9

M1 - 9

ER -