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Activation of polymorphonuclear neutrophils in patients with impaired left ventricular function.

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Activation of polymorphonuclear neutrophils in patients with impaired left ventricular function. / Rudolph, Volker; Rudolph, Tanja Katharina; Hennings, J Christopher; Blankenberg, Stefan; Schnabel, Renate; Steven, Daniel; Haddad, Munif; Knittel, Katharina; Wende, Silke; Wenzel, Jürgen; Münzel, Thomas; Heitzer, Thomas; Meinertz, Thomas; Hübner, Christian; Baldus, Stephan.

in: FREE RADICAL BIO MED, Jahrgang 43, Nr. 8, 8, 2007, S. 1189-1196.

Publikationen: SCORING: Beitrag in Fachzeitschrift/Zeitung › SCORING: Zeitschriftenaufsatz › Forschung › Begutachtung

Harvard

Rudolph, V, Rudolph, TK, Hennings, JC, Blankenberg, S, Schnabel, R, Steven, D, Haddad, M, Knittel, K, Wende, S, Wenzel, J, Münzel, T, Heitzer, T, Meinertz, T, Hübner, C & Baldus, S 2007, 'Activation of polymorphonuclear neutrophils in patients with impaired left ventricular function.', FREE RADICAL BIO MED, Jg. 43, Nr. 8, 8, S. 1189-1196. <http://www.ncbi.nlm.nih.gov/pubmed/17854714?dopt=Citation>

APA

Rudolph, V., Rudolph, T. K., Hennings, J. C., Blankenberg, S., Schnabel, R., Steven, D., Haddad, M., Knittel, K., Wende, S., Wenzel, J., Münzel, T., Heitzer, T., Meinertz, T., Hübner, C., & Baldus, S. (2007). Activation of polymorphonuclear neutrophils in patients with impaired left ventricular function. FREE RADICAL BIO MED, 43(8), 1189-1196. [8]. http://www.ncbi.nlm.nih.gov/pubmed/17854714?dopt=Citation

Vancouver

Rudolph V, Rudolph TK, Hennings JC, Blankenberg S, Schnabel R, Steven D et al. Activation of polymorphonuclear neutrophils in patients with impaired left ventricular function. FREE RADICAL BIO MED. 2007;43(8):1189-1196. 8.

Bibtex

@article{c24304334b154676b64d0c60a42ad069,
title = "Activation of polymorphonuclear neutrophils in patients with impaired left ventricular function.",
abstract = "Activation of leukocytes and in particular polymorphonuclear neutrophils (PMN) has emerged as a critical confounder in the pathophysiology of cardiovascular disease: Myeloperoxidase (MPO), one of the principal proteins hosted in and secreted by activated PMN, has been mechanistically linked to endothelial and left ventricular (LV) dysfunction in rodent models of sepsis and ischemic cardiomyopathy. Whether PMN activation is also overt in patients with LV dysfunction of ischemic and nonischemic origin, however, remains elusive. Prospectively, 447 consecutive, stable outpatients were included in this single-center study. In 113 patients with impaired left ventricular function (ejection fraction",
author = "Volker Rudolph and Rudolph, {Tanja Katharina} and Hennings, {J Christopher} and Stefan Blankenberg and Renate Schnabel and Daniel Steven and Munif Haddad and Katharina Knittel and Silke Wende and J{\"u}rgen Wenzel and Thomas M{\"u}nzel and Thomas Heitzer and Thomas Meinertz and Christian H{\"u}bner and Stephan Baldus",
year = "2007",
language = "Deutsch",
volume = "43",
pages = "1189--1196",
journal = "FREE RADICAL BIO MED",
issn = "0891-5849",
publisher = "Elsevier Inc.",
number = "8",

}

RIS

TY - JOUR

T1 - Activation of polymorphonuclear neutrophils in patients with impaired left ventricular function.

AU - Rudolph, Volker

AU - Rudolph, Tanja Katharina

AU - Hennings, J Christopher

AU - Blankenberg, Stefan

AU - Schnabel, Renate

AU - Steven, Daniel

AU - Haddad, Munif

AU - Knittel, Katharina

AU - Wende, Silke

AU - Wenzel, Jürgen

AU - Münzel, Thomas

AU - Heitzer, Thomas

AU - Meinertz, Thomas

AU - Hübner, Christian

AU - Baldus, Stephan

PY - 2007

Y1 - 2007

N2 - Activation of leukocytes and in particular polymorphonuclear neutrophils (PMN) has emerged as a critical confounder in the pathophysiology of cardiovascular disease: Myeloperoxidase (MPO), one of the principal proteins hosted in and secreted by activated PMN, has been mechanistically linked to endothelial and left ventricular (LV) dysfunction in rodent models of sepsis and ischemic cardiomyopathy. Whether PMN activation is also overt in patients with LV dysfunction of ischemic and nonischemic origin, however, remains elusive. Prospectively, 447 consecutive, stable outpatients were included in this single-center study. In 113 patients with impaired left ventricular function (ejection fraction

AB - Activation of leukocytes and in particular polymorphonuclear neutrophils (PMN) has emerged as a critical confounder in the pathophysiology of cardiovascular disease: Myeloperoxidase (MPO), one of the principal proteins hosted in and secreted by activated PMN, has been mechanistically linked to endothelial and left ventricular (LV) dysfunction in rodent models of sepsis and ischemic cardiomyopathy. Whether PMN activation is also overt in patients with LV dysfunction of ischemic and nonischemic origin, however, remains elusive. Prospectively, 447 consecutive, stable outpatients were included in this single-center study. In 113 patients with impaired left ventricular function (ejection fraction

M3 - SCORING: Zeitschriftenaufsatz

VL - 43

SP - 1189

EP - 1196

JO - FREE RADICAL BIO MED

JF - FREE RADICAL BIO MED

SN - 0891-5849

IS - 8

M1 - 8

ER -