Surprising Hyperkalemia of 10.2 mmol/L in a Patient with Hyperglycemia: A Case Report
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Surprising Hyperkalemia of 10.2 mmol/L in a Patient with Hyperglycemia: A Case Report. / Czogalla, Jan; Tariparast, Pischtaz Adel; Huber, Tobias B; Janneck, Matthias; Grahammer, Florian.
In: CASE REP NEPHROL DIA, Vol. 11, No. 1, 09.04.2021, p. 69-77.Research output: SCORING: Contribution to journal › SCORING: Journal article › Research › peer-review
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TY - JOUR
T1 - Surprising Hyperkalemia of 10.2 mmol/L in a Patient with Hyperglycemia: A Case Report
AU - Czogalla, Jan
AU - Tariparast, Pischtaz Adel
AU - Huber, Tobias B
AU - Janneck, Matthias
AU - Grahammer, Florian
N1 - Copyright © 2021 by S. Karger AG, Basel.
PY - 2021/4/9
Y1 - 2021/4/9
N2 - Hyperkalemia is a life-threatening condition potentially leading to cardiac arrest. Here, we report a case of surprising severe hyperkalemia of 10.2 mmol/L in a diabetic patient with previously normal kidney function presenting without discernible clinical symptoms to our emergency department. The patient was admitted because of hyperglycemia of 32.8 mmol/L, which was detected during daily testing in her nursing home. The hyperkalemia was caused by prerenal failure due to hyperglycemic polyuria which led to volume depletion, and worsened by a combination of potassium-sparing drugs and potassium supplementation. The patient was treated conservatively. Eighteen hours later, the serum potassium concentration was 4.6 mmol/L. The patient could be released 6 days later. To our knowledge, this is the highest described hyperkalemia treated conservatively and survived without cardiopulmonary resuscitation.
AB - Hyperkalemia is a life-threatening condition potentially leading to cardiac arrest. Here, we report a case of surprising severe hyperkalemia of 10.2 mmol/L in a diabetic patient with previously normal kidney function presenting without discernible clinical symptoms to our emergency department. The patient was admitted because of hyperglycemia of 32.8 mmol/L, which was detected during daily testing in her nursing home. The hyperkalemia was caused by prerenal failure due to hyperglycemic polyuria which led to volume depletion, and worsened by a combination of potassium-sparing drugs and potassium supplementation. The patient was treated conservatively. Eighteen hours later, the serum potassium concentration was 4.6 mmol/L. The patient could be released 6 days later. To our knowledge, this is the highest described hyperkalemia treated conservatively and survived without cardiopulmonary resuscitation.
U2 - 10.1159/000512590
DO - 10.1159/000512590
M3 - SCORING: Journal article
C2 - 33829044
VL - 11
SP - 69
EP - 77
JO - CASE REP NEPHROL DIA
JF - CASE REP NEPHROL DIA
SN - 2296-9705
IS - 1
ER -
