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[Oxidative stress at the retinal pigment epithelium - experimental implications for protection]

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[Oxidative stress at the retinal pigment epithelium - experimental implications for protection]. / Zeitz, Oliver; Berna-Thill, Michelle; Bartsch, Udo; Strauss, Olaf; Richard, Gisbert.

In: KLIN MONATSBL AUGENH, Vol. 226, No. 1, 1, 2009, p. 27-30.

Research output: SCORING: Contribution to journal › SCORING: Journal article › Research › peer-review

Harvard

Zeitz, O, Berna-Thill, M, Bartsch, U, Strauss, O & Richard, G 2009, '[Oxidative stress at the retinal pigment epithelium - experimental implications for protection]', KLIN MONATSBL AUGENH, vol. 226, no. 1, 1, pp. 27-30. <http://www.ncbi.nlm.nih.gov/pubmed/19173160?dopt=Citation>

APA

Zeitz, O., Berna-Thill, M., Bartsch, U., Strauss, O., & Richard, G. (2009). [Oxidative stress at the retinal pigment epithelium - experimental implications for protection]. KLIN MONATSBL AUGENH, 226(1), 27-30. [1]. http://www.ncbi.nlm.nih.gov/pubmed/19173160?dopt=Citation

Vancouver

Zeitz O, Berna-Thill M, Bartsch U, Strauss O, Richard G. [Oxidative stress at the retinal pigment epithelium - experimental implications for protection]. KLIN MONATSBL AUGENH. 2009;226(1):27-30. 1.

Bibtex

@article{544f3a0ea1124feeb5e72a479e762b2b,
title = "[Oxidative stress at the retinal pigment epithelium - experimental implications for protection]",
abstract = "Oxidative stress at the retinal pigment epithelium (RPE) is involved in the pathophysiology of age-related macula degeneration (ARMD). Observations on a clinical or laboratory level have revealed that supplementation of antioxidative scavengers failed in many cases. A potential therapeutic target is the cellular signal transduction cascade initiated by oxidative stress which results, e. g., in altered expression of pro- and antiagiogenic factors as well as induction of apoptosis. This review summarises the current literature on cellular effects of free radicals and deduces potential therapeutic approaches to protect the RPE from oxidative damage.",
author = "Oliver Zeitz and Michelle Berna-Thill and Udo Bartsch and Olaf Strauss and Gisbert Richard",
year = "2009",
language = "Deutsch",
volume = "226",
pages = "27--30",
journal = "KLIN MONATSBL AUGENH",
issn = "0023-2165",
publisher = "Ferdinand Enke Verlag",
number = "1",

}

RIS

TY - JOUR

T1 - [Oxidative stress at the retinal pigment epithelium - experimental implications for protection]

AU - Zeitz, Oliver

AU - Berna-Thill, Michelle

AU - Bartsch, Udo

AU - Strauss, Olaf

AU - Richard, Gisbert

PY - 2009

Y1 - 2009

N2 - Oxidative stress at the retinal pigment epithelium (RPE) is involved in the pathophysiology of age-related macula degeneration (ARMD). Observations on a clinical or laboratory level have revealed that supplementation of antioxidative scavengers failed in many cases. A potential therapeutic target is the cellular signal transduction cascade initiated by oxidative stress which results, e. g., in altered expression of pro- and antiagiogenic factors as well as induction of apoptosis. This review summarises the current literature on cellular effects of free radicals and deduces potential therapeutic approaches to protect the RPE from oxidative damage.

AB - Oxidative stress at the retinal pigment epithelium (RPE) is involved in the pathophysiology of age-related macula degeneration (ARMD). Observations on a clinical or laboratory level have revealed that supplementation of antioxidative scavengers failed in many cases. A potential therapeutic target is the cellular signal transduction cascade initiated by oxidative stress which results, e. g., in altered expression of pro- and antiagiogenic factors as well as induction of apoptosis. This review summarises the current literature on cellular effects of free radicals and deduces potential therapeutic approaches to protect the RPE from oxidative damage.

M3 - SCORING: Zeitschriftenaufsatz

VL - 226

SP - 27

EP - 30

JO - KLIN MONATSBL AUGENH

JF - KLIN MONATSBL AUGENH

SN - 0023-2165

IS - 1

M1 - 1

ER -