Oculogyric crises: Etiology, pathophysiology and therapeutic approaches

Ewgenia Barow; Susanne A Schneider; Kailash P Bhatia; Christos Ganos

doi:10.1016/j.parkreldis.2016.11.012

Oculogyric crises: Etiology, pathophysiology and therapeutic approaches

Standard

Oculogyric crises: Etiology, pathophysiology and therapeutic approaches. / Barow, Ewgenia; Schneider, Susanne A; Bhatia, Kailash P; Ganos, Christos.

In: PARKINSONISM RELAT D, Vol. 36, 03.2017, p. 3-9.

Research output: SCORING: Contribution to journal › SCORING: Journal article › Research › peer-review

Harvard

Barow, E, Schneider, SA, Bhatia, KP & Ganos, C 2017, 'Oculogyric crises: Etiology, pathophysiology and therapeutic approaches', PARKINSONISM RELAT D, vol. 36, pp. 3-9. https://doi.org/10.1016/j.parkreldis.2016.11.012

APA

Barow, E., Schneider, S. A., Bhatia, K. P., & Ganos, C. (2017). Oculogyric crises: Etiology, pathophysiology and therapeutic approaches. PARKINSONISM RELAT D, 36, 3-9. https://doi.org/10.1016/j.parkreldis.2016.11.012

Vancouver

Barow E, Schneider SA, Bhatia KP, Ganos C. Oculogyric crises: Etiology, pathophysiology and therapeutic approaches. PARKINSONISM RELAT D. 2017 Mar;36:3-9. https://doi.org/10.1016/j.parkreldis.2016.11.012

Bibtex

@article{0359b83b67a34e5095a88e9d11399000,

title = "Oculogyric crises: Etiology, pathophysiology and therapeutic approaches",

abstract = "Oculogyric crisis (OGC) describes the clinical phenomenon of sustained dystonic, conjugate and typically upward deviation of the eyes lasting from seconds to hours. It was initially observed in patients with postencephalitic parkinsonism, but since then a number of conditions have been associated with OGC. These include drug-induced reactions, hereditary and sporadic movement disorders, and focal brain lesions. Here, we systematically review the literature and discuss the spectrum of disorders associated with OGC in order to aid clinicians place this rare but distinctive clinical sign into the appropriate diagnostic context. We also provide a brief synthesis of putative pathophysiological mechanisms, as well as therapeutic recommendations based on the literature and our own experience.",

author = "Ewgenia Barow and Schneider, {Susanne A} and Bhatia, {Kailash P} and Christos Ganos",

year = "2017",

month = mar,

doi = "10.1016/j.parkreldis.2016.11.012",

language = "English",

volume = "36",

pages = "3--9",

journal = "PARKINSONISM RELAT D",

issn = "1353-8020",

publisher = "Elsevier BV",

}

RIS

TY - JOUR

T1 - Oculogyric crises: Etiology, pathophysiology and therapeutic approaches

AU - Barow, Ewgenia

AU - Schneider, Susanne A

AU - Bhatia, Kailash P

AU - Ganos, Christos

PY - 2017/3

Y1 - 2017/3

N2 - Oculogyric crisis (OGC) describes the clinical phenomenon of sustained dystonic, conjugate and typically upward deviation of the eyes lasting from seconds to hours. It was initially observed in patients with postencephalitic parkinsonism, but since then a number of conditions have been associated with OGC. These include drug-induced reactions, hereditary and sporadic movement disorders, and focal brain lesions. Here, we systematically review the literature and discuss the spectrum of disorders associated with OGC in order to aid clinicians place this rare but distinctive clinical sign into the appropriate diagnostic context. We also provide a brief synthesis of putative pathophysiological mechanisms, as well as therapeutic recommendations based on the literature and our own experience.

AB - Oculogyric crisis (OGC) describes the clinical phenomenon of sustained dystonic, conjugate and typically upward deviation of the eyes lasting from seconds to hours. It was initially observed in patients with postencephalitic parkinsonism, but since then a number of conditions have been associated with OGC. These include drug-induced reactions, hereditary and sporadic movement disorders, and focal brain lesions. Here, we systematically review the literature and discuss the spectrum of disorders associated with OGC in order to aid clinicians place this rare but distinctive clinical sign into the appropriate diagnostic context. We also provide a brief synthesis of putative pathophysiological mechanisms, as well as therapeutic recommendations based on the literature and our own experience.

U2 - 10.1016/j.parkreldis.2016.11.012

DO - 10.1016/j.parkreldis.2016.11.012

M3 - SCORING: Journal article

C2 - 27964831

VL - 36

SP - 3

EP - 9

JO - PARKINSONISM RELAT D

JF - PARKINSONISM RELAT D

SN - 1353-8020

ER -