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Neurobiological Basis of Hypersexuality

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Neurobiological Basis of Hypersexuality. / Kühn, S; Gallinat, J.

Imaging the addicted brain. ed. / NM Zahr; ET Peterson. Vol. 129 1. ed. 2016. p. 67-83 Chapter 3 (International Review of Neurobiology).

Research output: SCORING: Contribution to book/anthology › SCORING: Contribution to collected editions/anthologies › Research › peer-review

Harvard

Kühn, S & Gallinat, J 2016, Neurobiological Basis of Hypersexuality. in NM Zahr & ET Peterson (eds), Imaging the addicted brain. 1 edn, vol. 129, Chapter 3, International Review of Neurobiology, pp. 67-83. https://doi.org/10.1016/bs.irn.2016.04.002

APA

Kühn, S., & Gallinat, J. (2016). Neurobiological Basis of Hypersexuality. In NM. Zahr, & ET. Peterson (Eds.), Imaging the addicted brain (1 ed., Vol. 129, pp. 67-83). [Chapter 3] (International Review of Neurobiology). https://doi.org/10.1016/bs.irn.2016.04.002

Vancouver

Kühn S, Gallinat J. Neurobiological Basis of Hypersexuality. In Zahr NM, Peterson ET, editors, Imaging the addicted brain. 1 ed. Vol. 129. 2016. p. 67-83. Chapter 3. (International Review of Neurobiology). https://doi.org/10.1016/bs.irn.2016.04.002

Bibtex

@inbook{f74aa2bbff6d43219bf5aabcf64de614,
title = "Neurobiological Basis of Hypersexuality",
abstract = "Until now, hypersexuality has not found entry into the common diagnostic classification systems. However it is a frequently discussed phenomenon consisting of excessive sexual appetite that is maladaptive for the individual. Initial studies investigated the neurobiological underpinnings of hypersexuality, but current literature is still insufficient to draw unequivocal conclusions. In the present review, we summarize and discuss findings from various perspectives: neuroimaging and lesion studies, studies on other neurological disorders that are sometimes accompanied by hypersexuality, neuropharmacological evidence, genetic as well as animal studies. Taken together, the evidence seems to imply that alterations in the frontal lobe, amygdala, hippocampus, hypothalamus, septum, and brain regions that process reward play a prominent role in the emergence of hypersexuality. Genetic studies and neuropharmacological treatment approaches point at an involvement of the dopaminergic system.",
keywords = "Journal Article",
author = "S K{\"u}hn and J Gallinat",
note = "{\textcopyright} 2016 Elsevier Inc. All rights reserved.",
year = "2016",
doi = "10.1016/bs.irn.2016.04.002",
language = "English",
isbn = "978-0-12-803914-4",
volume = "129",
series = "International Review of Neurobiology",
publisher = "ELSEVIER ACADEMIC PRESS INC",
pages = "67--83",
editor = "NM Zahr and ET Peterson",
booktitle = "Imaging the addicted brain",
edition = "1",

}

RIS

TY - CHAP

T1 - Neurobiological Basis of Hypersexuality

AU - Kühn, S

AU - Gallinat, J

N1 - © 2016 Elsevier Inc. All rights reserved.

PY - 2016

Y1 - 2016

N2 - Until now, hypersexuality has not found entry into the common diagnostic classification systems. However it is a frequently discussed phenomenon consisting of excessive sexual appetite that is maladaptive for the individual. Initial studies investigated the neurobiological underpinnings of hypersexuality, but current literature is still insufficient to draw unequivocal conclusions. In the present review, we summarize and discuss findings from various perspectives: neuroimaging and lesion studies, studies on other neurological disorders that are sometimes accompanied by hypersexuality, neuropharmacological evidence, genetic as well as animal studies. Taken together, the evidence seems to imply that alterations in the frontal lobe, amygdala, hippocampus, hypothalamus, septum, and brain regions that process reward play a prominent role in the emergence of hypersexuality. Genetic studies and neuropharmacological treatment approaches point at an involvement of the dopaminergic system.

AB - Until now, hypersexuality has not found entry into the common diagnostic classification systems. However it is a frequently discussed phenomenon consisting of excessive sexual appetite that is maladaptive for the individual. Initial studies investigated the neurobiological underpinnings of hypersexuality, but current literature is still insufficient to draw unequivocal conclusions. In the present review, we summarize and discuss findings from various perspectives: neuroimaging and lesion studies, studies on other neurological disorders that are sometimes accompanied by hypersexuality, neuropharmacological evidence, genetic as well as animal studies. Taken together, the evidence seems to imply that alterations in the frontal lobe, amygdala, hippocampus, hypothalamus, septum, and brain regions that process reward play a prominent role in the emergence of hypersexuality. Genetic studies and neuropharmacological treatment approaches point at an involvement of the dopaminergic system.

KW - Journal Article

U2 - 10.1016/bs.irn.2016.04.002

DO - 10.1016/bs.irn.2016.04.002

M3 - SCORING: Contribution to collected editions/anthologies

C2 - 27503448

SN - 978-0-12-803914-4

VL - 129

T3 - International Review of Neurobiology

SP - 67

EP - 83

BT - Imaging the addicted brain

A2 - Zahr, NM

A2 - Peterson, ET

ER -