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Dynamic development of glucocorticoid resistance during autoimmune neuroinflammation.

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Dynamic development of glucocorticoid resistance during autoimmune neuroinflammation. / Gold, Stefan; Sasidhar, Manda V; Lagishetty, Venu; Spence, Rory D; Umeda, Elizabeth; Ziehn, Marina O; Krieger, Thorsten; Schulz, Karl-Heinz; Heesen, Christoph; Hewison, Martin; Voskuhl, Rhonda R.

In: J CLIN ENDOCR METAB, Vol. 97, No. 8, 8, 2012, p. 1402-1410.

Research output: SCORING: Contribution to journal › SCORING: Journal article › Research › peer-review

Harvard

Gold, S, Sasidhar, MV, Lagishetty, V, Spence, RD, Umeda, E, Ziehn, MO, Krieger, T, Schulz, K-H, Heesen, C, Hewison, M & Voskuhl, RR 2012, 'Dynamic development of glucocorticoid resistance during autoimmune neuroinflammation.', J CLIN ENDOCR METAB, vol. 97, no. 8, 8, pp. 1402-1410. <http://www.ncbi.nlm.nih.gov/pubmed/22659246?dopt=Citation>

APA

Gold, S., Sasidhar, M. V., Lagishetty, V., Spence, R. D., Umeda, E., Ziehn, M. O., Krieger, T., Schulz, K-H., Heesen, C., Hewison, M., & Voskuhl, R. R. (2012). Dynamic development of glucocorticoid resistance during autoimmune neuroinflammation. J CLIN ENDOCR METAB, 97(8), 1402-1410. [8]. http://www.ncbi.nlm.nih.gov/pubmed/22659246?dopt=Citation

Vancouver

Gold S, Sasidhar MV, Lagishetty V, Spence RD, Umeda E, Ziehn MO et al. Dynamic development of glucocorticoid resistance during autoimmune neuroinflammation. J CLIN ENDOCR METAB. 2012;97(8):1402-1410. 8.

Bibtex

@article{c7a36b16b8ad409aaad8cfd19e564dbc,
title = "Dynamic development of glucocorticoid resistance during autoimmune neuroinflammation.",
abstract = "Glucocorticoids (GC) are powerful endogenous and therapeutic modulators of inflammation and play a critical role for controlling autoimmunity. GC resistance can be seen in patients with cell-mediated autoimmune disorders, but it is unknown whether this represents a stable trait or a state.",
keywords = "Animals, Humans, Male, Female, Cross-Sectional Studies, Mice, Mice, Inbred C57BL, Apoptosis/drug effects, Drug Resistance, T-Lymphocytes/immunology, Peptide Fragments/immunology, Autoantigens/immunology, Encephalomyelitis, Autoimmune, Experimental/*drug therapy/immunology, Glucocorticoids/*therapeutic use, Glycoproteins/immunology, Multiple Sclerosis/*drug therapy/immunology, Myelin-Oligodendrocyte Glycoprotein, Receptors, Glucocorticoid/genetics, Animals, Humans, Male, Female, Cross-Sectional Studies, Mice, Mice, Inbred C57BL, Apoptosis/drug effects, Drug Resistance, T-Lymphocytes/immunology, Peptide Fragments/immunology, Autoantigens/immunology, Encephalomyelitis, Autoimmune, Experimental/*drug therapy/immunology, Glucocorticoids/*therapeutic use, Glycoproteins/immunology, Multiple Sclerosis/*drug therapy/immunology, Myelin-Oligodendrocyte Glycoprotein, Receptors, Glucocorticoid/genetics",
author = "Stefan Gold and Sasidhar, {Manda V} and Venu Lagishetty and Spence, {Rory D} and Elizabeth Umeda and Ziehn, {Marina O} and Thorsten Krieger and Karl-Heinz Schulz and Christoph Heesen and Martin Hewison and Voskuhl, {Rhonda R}",
year = "2012",
language = "English",
volume = "97",
pages = "1402--1410",
journal = "J CLIN ENDOCR METAB",
issn = "0021-972X",
publisher = "The Endocrine Society",
number = "8",

}

RIS

TY - JOUR

T1 - Dynamic development of glucocorticoid resistance during autoimmune neuroinflammation.

AU - Gold, Stefan

AU - Sasidhar, Manda V

AU - Lagishetty, Venu

AU - Spence, Rory D

AU - Umeda, Elizabeth

AU - Ziehn, Marina O

AU - Krieger, Thorsten

AU - Schulz, Karl-Heinz

AU - Heesen, Christoph

AU - Hewison, Martin

AU - Voskuhl, Rhonda R

PY - 2012

Y1 - 2012

N2 - Glucocorticoids (GC) are powerful endogenous and therapeutic modulators of inflammation and play a critical role for controlling autoimmunity. GC resistance can be seen in patients with cell-mediated autoimmune disorders, but it is unknown whether this represents a stable trait or a state.

AB - Glucocorticoids (GC) are powerful endogenous and therapeutic modulators of inflammation and play a critical role for controlling autoimmunity. GC resistance can be seen in patients with cell-mediated autoimmune disorders, but it is unknown whether this represents a stable trait or a state.

KW - Animals

KW - Humans

KW - Male

KW - Female

KW - Cross-Sectional Studies

KW - Mice

KW - Mice, Inbred C57BL

KW - Apoptosis/drug effects

KW - Drug Resistance

KW - T-Lymphocytes/immunology

KW - Peptide Fragments/immunology

KW - Autoantigens/immunology

KW - Encephalomyelitis, Autoimmune, Experimental/drug therapy/immunology

KW - Glucocorticoids/therapeutic use

KW - Glycoproteins/immunology

KW - Multiple Sclerosis/drug therapy/immunology

KW - Myelin-Oligodendrocyte Glycoprotein

KW - Receptors, Glucocorticoid/genetics

KW - Animals

KW - Humans

KW - Male

KW - Female

KW - Cross-Sectional Studies

KW - Mice

KW - Mice, Inbred C57BL

KW - Apoptosis/drug effects

KW - Drug Resistance

KW - T-Lymphocytes/immunology

KW - Peptide Fragments/immunology

KW - Autoantigens/immunology

KW - Encephalomyelitis, Autoimmune, Experimental/drug therapy/immunology

KW - Glucocorticoids/therapeutic use

KW - Glycoproteins/immunology

KW - Multiple Sclerosis/drug therapy/immunology

KW - Myelin-Oligodendrocyte Glycoprotein

KW - Receptors, Glucocorticoid/genetics

M3 - SCORING: Journal article

VL - 97

SP - 1402

EP - 1410

JO - J CLIN ENDOCR METAB

JF - J CLIN ENDOCR METAB

SN - 0021-972X

IS - 8

M1 - 8

ER -