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Chronic liver inflammation dominated by interferon-γ can prevent hepatocarcinogenesis.

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Chronic liver inflammation dominated by interferon-γ can prevent hepatocarcinogenesis. / Schrader, Jörg; Herkel, Johannes.

In: ONCOIMMUNOLOGY, Vol. 1, No. 2, 2, 2012, p. 222-223.

Research output: SCORING: Contribution to journal › SCORING: Journal article › Research › peer-review

Harvard

Schrader, J & Herkel, J 2012, 'Chronic liver inflammation dominated by interferon-γ can prevent hepatocarcinogenesis.', ONCOIMMUNOLOGY, vol. 1, no. 2, 2, pp. 222-223. <http://www.ncbi.nlm.nih.gov/pubmed/22720249?dopt=Citation>

APA

Schrader, J., & Herkel, J. (2012). Chronic liver inflammation dominated by interferon-γ can prevent hepatocarcinogenesis. ONCOIMMUNOLOGY, 1(2), 222-223. [2]. http://www.ncbi.nlm.nih.gov/pubmed/22720249?dopt=Citation

Vancouver

Schrader J, Herkel J. Chronic liver inflammation dominated by interferon-γ can prevent hepatocarcinogenesis. ONCOIMMUNOLOGY. 2012;1(2):222-223. 2.

Bibtex

@article{2fb67e770de44d4bb7eb2ec5c3f2b9ce,
title = "Chronic liver inflammation dominated by interferon-γ can prevent hepatocarcinogenesis.",
abstract = "Inflammation is a major stimulus for carcinogenesis; however inflammation can also inhibit tumor growth and deplete malignant cells. The differences between cancer-promoting and cancer-inhibitory inflammation are not clear. We identified Interferon-? as a major mediator of cancer-inhibitory inflammation that promotes anti-cancer immunity in the liver and sensitizes malignant hepatocytes for apoptosis.",
author = "J{\"o}rg Schrader and Johannes Herkel",
year = "2012",
language = "English",
volume = "1",
pages = "222--223",
journal = "ONCOIMMUNOLOGY",
issn = "2162-402X",
publisher = "Taylor & Francis",
number = "2",

}

RIS

TY - JOUR

T1 - Chronic liver inflammation dominated by interferon-γ can prevent hepatocarcinogenesis.

AU - Schrader, Jörg

AU - Herkel, Johannes

PY - 2012

Y1 - 2012

N2 - Inflammation is a major stimulus for carcinogenesis; however inflammation can also inhibit tumor growth and deplete malignant cells. The differences between cancer-promoting and cancer-inhibitory inflammation are not clear. We identified Interferon-? as a major mediator of cancer-inhibitory inflammation that promotes anti-cancer immunity in the liver and sensitizes malignant hepatocytes for apoptosis.

AB - Inflammation is a major stimulus for carcinogenesis; however inflammation can also inhibit tumor growth and deplete malignant cells. The differences between cancer-promoting and cancer-inhibitory inflammation are not clear. We identified Interferon-? as a major mediator of cancer-inhibitory inflammation that promotes anti-cancer immunity in the liver and sensitizes malignant hepatocytes for apoptosis.

M3 - SCORING: Journal article

VL - 1

SP - 222

EP - 223

JO - ONCOIMMUNOLOGY

JF - ONCOIMMUNOLOGY

SN - 2162-402X

IS - 2

M1 - 2

ER -