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Cellular Contributors to Bone Homeostasis

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Cellular Contributors to Bone Homeostasis. / Rauner, Martina; Jähn, Katharina; Hemmatian, Haniyeh; Colditz, Juliane; Goettsch, Claudia.

Cardiovascular Calcification and Bone Mineralization. ed. / Elena Aikawa; Joshua D. Hutcheson. Cham : Springer International Publishing, 2020. p. 333-371 (Contemporary Cardiology (CONCARD)).

Research output: SCORING: Contribution to book/anthology › Chapter › Research › peer-review

Harvard

Rauner, M, Jähn, K, Hemmatian, H, Colditz, J & Goettsch, C 2020, Cellular Contributors to Bone Homeostasis. in E Aikawa & JD Hutcheson (eds), Cardiovascular Calcification and Bone Mineralization. Contemporary Cardiology (CONCARD), Springer International Publishing, Cham, pp. 333-371. https://doi.org/10.1007/978-3-030-46725-8_16

APA

Rauner, M., Jähn, K., Hemmatian, H., Colditz, J., & Goettsch, C. (2020). Cellular Contributors to Bone Homeostasis. In E. Aikawa, & J. D. Hutcheson (Eds.), Cardiovascular Calcification and Bone Mineralization (pp. 333-371). (Contemporary Cardiology (CONCARD)). Springer International Publishing. https://doi.org/10.1007/978-3-030-46725-8_16

Vancouver

Rauner M, Jähn K, Hemmatian H, Colditz J, Goettsch C. Cellular Contributors to Bone Homeostasis. In Aikawa E, Hutcheson JD, editors, Cardiovascular Calcification and Bone Mineralization. Cham: Springer International Publishing. 2020. p. 333-371. (Contemporary Cardiology (CONCARD)). https://doi.org/10.1007/978-3-030-46725-8_16

Bibtex

@inbook{de6cee2ae5714bab8afe8499262e21f2,
title = "Cellular Contributors to Bone Homeostasis",
abstract = "Bone is a complex and dynamic tissue that, in addition to its biomechanical properties, harbors stem cells and precursor cells of the hematopoietic and immune system. The skeleton undergoes continuous remodeling processes to maintain proper bone homeostasis. The equilibrium and coupling of bone resorption with bone formation is fine-tuned by the controlled actions of distinct cell types: bone-forming osteoblasts, bone-resorbing osteoclasts, and osteocytes as key communicators. This ensures proper bone mass and quality. Under pathophysiological conditions, the impairment of these mechanisms triggers bone remodeling dysregulation, causing loss in bone mass and/or alteration in bone microarchitecture.",
author = "Martina Rauner and Katharina J{\"a}hn and Haniyeh Hemmatian and Juliane Colditz and Claudia Goettsch",
year = "2020",
doi = "10.1007/978-3-030-46725-8_16",
language = "Deutsch",
isbn = "978-3-030-46725-8",
series = "Contemporary Cardiology (CONCARD)",
publisher = "Springer International Publishing",
pages = "333--371",
editor = "Elena Aikawa and Hutcheson, {Joshua D.}",
booktitle = "Cardiovascular Calcification and Bone Mineralization",
address = "Schweiz",

}

RIS

TY - CHAP

T1 - Cellular Contributors to Bone Homeostasis

AU - Rauner, Martina

AU - Jähn, Katharina

AU - Hemmatian, Haniyeh

AU - Colditz, Juliane

AU - Goettsch, Claudia

PY - 2020

Y1 - 2020

N2 - Bone is a complex and dynamic tissue that, in addition to its biomechanical properties, harbors stem cells and precursor cells of the hematopoietic and immune system. The skeleton undergoes continuous remodeling processes to maintain proper bone homeostasis. The equilibrium and coupling of bone resorption with bone formation is fine-tuned by the controlled actions of distinct cell types: bone-forming osteoblasts, bone-resorbing osteoclasts, and osteocytes as key communicators. This ensures proper bone mass and quality. Under pathophysiological conditions, the impairment of these mechanisms triggers bone remodeling dysregulation, causing loss in bone mass and/or alteration in bone microarchitecture.

AB - Bone is a complex and dynamic tissue that, in addition to its biomechanical properties, harbors stem cells and precursor cells of the hematopoietic and immune system. The skeleton undergoes continuous remodeling processes to maintain proper bone homeostasis. The equilibrium and coupling of bone resorption with bone formation is fine-tuned by the controlled actions of distinct cell types: bone-forming osteoblasts, bone-resorbing osteoclasts, and osteocytes as key communicators. This ensures proper bone mass and quality. Under pathophysiological conditions, the impairment of these mechanisms triggers bone remodeling dysregulation, causing loss in bone mass and/or alteration in bone microarchitecture.

U2 - 10.1007/978-3-030-46725-8_16

DO - 10.1007/978-3-030-46725-8_16

M3 - Kapitel

SN - 978-3-030-46725-8

T3 - Contemporary Cardiology (CONCARD)

SP - 333

EP - 371

BT - Cardiovascular Calcification and Bone Mineralization

A2 - Aikawa, Elena

A2 - Hutcheson, Joshua D.

PB - Springer International Publishing

CY - Cham

ER -