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Role of sphenopalatine ganglion stimulation in cluster headache

Standard

Role of sphenopalatine ganglion stimulation in cluster headache. / Jürgens, Tim P; May, Arne.

in: CURR PAIN HEADACHE R, Jahrgang 18, Nr. 7, 01.07.2014, S. 433.

Publikationen: SCORING: Beitrag in Fachzeitschrift/Zeitung › SCORING: Zeitschriftenaufsatz › Forschung › Begutachtung

Harvard

Jürgens, TP & May, A 2014, 'Role of sphenopalatine ganglion stimulation in cluster headache', CURR PAIN HEADACHE R, Jg. 18, Nr. 7, S. 433. https://doi.org/10.1007/s11916-014-0433-4

APA

Jürgens, T. P., & May, A. (2014). Role of sphenopalatine ganglion stimulation in cluster headache. CURR PAIN HEADACHE R, 18(7), 433. https://doi.org/10.1007/s11916-014-0433-4

Vancouver

Jürgens TP, May A. Role of sphenopalatine ganglion stimulation in cluster headache. CURR PAIN HEADACHE R. 2014 Jul 1;18(7):433. https://doi.org/10.1007/s11916-014-0433-4

Bibtex

@article{8bd88a5d72864767b1f824f67c84fc61,
title = "Role of sphenopalatine ganglion stimulation in cluster headache",
abstract = "Cluster headache attacks are characterized by extreme unilateral pain mostly in the first trigeminal branch and an ipsilateral activation of the cranial parasympathetic system, pointing to a relevant role of the cranial parasympathetic system in the pathophysiology, and therapy of cluster headache. Based on animal experiments and several interventions of the sphenopalatine ganglion (such as an aesthetic or alcoholic blocks and radiofrequency ablation) in cluster headache patients, stimulation of the sphenopalatine ganglion (SPGS) as the major efferent peripheral parasympathetic structure was established with an encouraging abortive effect on acute attacks and a frequency reduction over time. In this review, the clinical data and potentially underlying pathophysiological concepts of SPGS are discussed in detail, which in brief point to a relevant role of the parasympathetic system both in the induction and termination of attacks.",
author = "J{\"u}rgens, {Tim P} and Arne May",
year = "2014",
month = jul,
day = "1",
doi = "10.1007/s11916-014-0433-4",
language = "English",
volume = "18",
pages = "433",
journal = "CURR PAIN HEADACHE R",
issn = "1531-3433",
publisher = "Current Science, Inc.",
number = "7",

}

RIS

TY - JOUR

T1 - Role of sphenopalatine ganglion stimulation in cluster headache

AU - Jürgens, Tim P

AU - May, Arne

PY - 2014/7/1

Y1 - 2014/7/1

N2 - Cluster headache attacks are characterized by extreme unilateral pain mostly in the first trigeminal branch and an ipsilateral activation of the cranial parasympathetic system, pointing to a relevant role of the cranial parasympathetic system in the pathophysiology, and therapy of cluster headache. Based on animal experiments and several interventions of the sphenopalatine ganglion (such as an aesthetic or alcoholic blocks and radiofrequency ablation) in cluster headache patients, stimulation of the sphenopalatine ganglion (SPGS) as the major efferent peripheral parasympathetic structure was established with an encouraging abortive effect on acute attacks and a frequency reduction over time. In this review, the clinical data and potentially underlying pathophysiological concepts of SPGS are discussed in detail, which in brief point to a relevant role of the parasympathetic system both in the induction and termination of attacks.

AB - Cluster headache attacks are characterized by extreme unilateral pain mostly in the first trigeminal branch and an ipsilateral activation of the cranial parasympathetic system, pointing to a relevant role of the cranial parasympathetic system in the pathophysiology, and therapy of cluster headache. Based on animal experiments and several interventions of the sphenopalatine ganglion (such as an aesthetic or alcoholic blocks and radiofrequency ablation) in cluster headache patients, stimulation of the sphenopalatine ganglion (SPGS) as the major efferent peripheral parasympathetic structure was established with an encouraging abortive effect on acute attacks and a frequency reduction over time. In this review, the clinical data and potentially underlying pathophysiological concepts of SPGS are discussed in detail, which in brief point to a relevant role of the parasympathetic system both in the induction and termination of attacks.

U2 - 10.1007/s11916-014-0433-4

DO - 10.1007/s11916-014-0433-4

M3 - SCORING: Journal article

C2 - 24880803

VL - 18

SP - 433

JO - CURR PAIN HEADACHE R

JF - CURR PAIN HEADACHE R

SN - 1531-3433

IS - 7

ER -