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It's not all about nephrin

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It's not all about nephrin. / Simons, M; Huber, T B.

in: KIDNEY INT, Jahrgang 73, Nr. 6, 03.2008, S. 671-3.

Publikationen: SCORING: Beitrag in Fachzeitschrift/Zeitung › SCORING: Zeitschriftenaufsatz › Forschung › Begutachtung

Harvard

Simons, M & Huber, TB 2008, 'It's not all about nephrin', KIDNEY INT, Jg. 73, Nr. 6, S. 671-3. https://doi.org/10.1038/sj.ki.5002798

APA

Simons, M., & Huber, T. B. (2008). It's not all about nephrin. KIDNEY INT, 73(6), 671-3. https://doi.org/10.1038/sj.ki.5002798

Vancouver

Simons M, Huber TB. It's not all about nephrin. KIDNEY INT. 2008 Mär;73(6):671-3. https://doi.org/10.1038/sj.ki.5002798

Bibtex

@article{e33b5ed5cefc405bb0d882603c8af8a6,
title = "It's not all about nephrin",
abstract = "Mutations in the NPHS1 gene cause congenital nephrotic syndrome of the Finnish type. The gene product nephrin is a structural component of the glomerular slit diaphragm formed by neighboring podocytes. Nephrin has also been suggested to be involved in signaling processes that are important for podocyte survival and differentiation. A new study by Don{\'e} et al. reports that the absence of nephrin leads to the lack of slit diaphragms but does not affect podocyte apoptosis and gene expression patterns.",
keywords = "Animals, Apoptosis, Disease Models, Animal, Gene Expression, Humans, Kidney Glomerulus, Membrane Proteins, Mice, Mice, Knockout, Nephrotic Syndrome, Podocytes, Comment, Journal Article",
author = "M Simons and Huber, {T B}",
year = "2008",
month = mar,
doi = "10.1038/sj.ki.5002798",
language = "English",
volume = "73",
pages = "671--3",
journal = "KIDNEY INT",
issn = "0085-2538",
publisher = "NATURE PUBLISHING GROUP",
number = "6",

}

RIS

TY - JOUR

T1 - It's not all about nephrin

AU - Simons, M

AU - Huber, T B

PY - 2008/3

Y1 - 2008/3

N2 - Mutations in the NPHS1 gene cause congenital nephrotic syndrome of the Finnish type. The gene product nephrin is a structural component of the glomerular slit diaphragm formed by neighboring podocytes. Nephrin has also been suggested to be involved in signaling processes that are important for podocyte survival and differentiation. A new study by Doné et al. reports that the absence of nephrin leads to the lack of slit diaphragms but does not affect podocyte apoptosis and gene expression patterns.

AB - Mutations in the NPHS1 gene cause congenital nephrotic syndrome of the Finnish type. The gene product nephrin is a structural component of the glomerular slit diaphragm formed by neighboring podocytes. Nephrin has also been suggested to be involved in signaling processes that are important for podocyte survival and differentiation. A new study by Doné et al. reports that the absence of nephrin leads to the lack of slit diaphragms but does not affect podocyte apoptosis and gene expression patterns.

KW - Animals

KW - Apoptosis

KW - Disease Models, Animal

KW - Gene Expression

KW - Humans

KW - Kidney Glomerulus

KW - Membrane Proteins

KW - Mice

KW - Mice, Knockout

KW - Nephrotic Syndrome

KW - Podocytes

KW - Comment

KW - Journal Article

U2 - 10.1038/sj.ki.5002798

DO - 10.1038/sj.ki.5002798

M3 - SCORING: Journal article

C2 - 18309348

VL - 73

SP - 671

EP - 673

JO - KIDNEY INT

JF - KIDNEY INT

SN - 0085-2538

IS - 6

ER -