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Implications of autophagy for glomerular aging and disease

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Implications of autophagy for glomerular aging and disease. / Weide, Thomas; Huber, Tobias B.

in: CELL TISSUE RES, Jahrgang 343, Nr. 3, 03.2011, S. 467-73.

Publikationen: SCORING: Beitrag in Fachzeitschrift/Zeitung › SCORING: Review › Forschung

Harvard

Weide, T & Huber, TB 2011, 'Implications of autophagy for glomerular aging and disease', CELL TISSUE RES, Jg. 343, Nr. 3, S. 467-73. https://doi.org/10.1007/s00441-010-1115-0

APA

Weide, T., & Huber, T. B. (2011). Implications of autophagy for glomerular aging and disease. CELL TISSUE RES, 343(3), 467-73. https://doi.org/10.1007/s00441-010-1115-0

Vancouver

Weide T, Huber TB. Implications of autophagy for glomerular aging and disease. CELL TISSUE RES. 2011 Mär;343(3):467-73. https://doi.org/10.1007/s00441-010-1115-0

Bibtex

@article{15d01261e0454bb691332fd98802b48b,
title = "Implications of autophagy for glomerular aging and disease",
abstract = "Glomerular diseases lead to a progressive decline in renal function and account for the vast majority of end-stage kidney diseases. Injury and loss of glomerular podocytes are common determining factors of glomerular disease progression and renal failure. Podocytes are a primary glomerular target of toxic, immune, metabolic, and oxidant stress, but little is known of the factors that counteract cellular stress signaling pathways. This review focuses on recent findings that identify autophagy as a critical homeostatic and quality control mechanism maintaining glomerular homeostasis.",
keywords = "Aging, Autophagy, Endosomes, Homeostasis, Humans, Kidney Diseases, Kidney Glomerulus, Lysosomes, TOR Serine-Threonine Kinases, Journal Article, Research Support, Non-U.S. Gov't, Review",
author = "Thomas Weide and Huber, {Tobias B}",
year = "2011",
month = mar,
doi = "10.1007/s00441-010-1115-0",
language = "English",
volume = "343",
pages = "467--73",
journal = "CELL TISSUE RES",
issn = "0302-766X",
publisher = "Springer",
number = "3",

}

RIS

TY - JOUR

T1 - Implications of autophagy for glomerular aging and disease

AU - Weide, Thomas

AU - Huber, Tobias B

PY - 2011/3

Y1 - 2011/3

N2 - Glomerular diseases lead to a progressive decline in renal function and account for the vast majority of end-stage kidney diseases. Injury and loss of glomerular podocytes are common determining factors of glomerular disease progression and renal failure. Podocytes are a primary glomerular target of toxic, immune, metabolic, and oxidant stress, but little is known of the factors that counteract cellular stress signaling pathways. This review focuses on recent findings that identify autophagy as a critical homeostatic and quality control mechanism maintaining glomerular homeostasis.

AB - Glomerular diseases lead to a progressive decline in renal function and account for the vast majority of end-stage kidney diseases. Injury and loss of glomerular podocytes are common determining factors of glomerular disease progression and renal failure. Podocytes are a primary glomerular target of toxic, immune, metabolic, and oxidant stress, but little is known of the factors that counteract cellular stress signaling pathways. This review focuses on recent findings that identify autophagy as a critical homeostatic and quality control mechanism maintaining glomerular homeostasis.

KW - Aging

KW - Autophagy

KW - Endosomes

KW - Homeostasis

KW - Humans

KW - Kidney Diseases

KW - Kidney Glomerulus

KW - Lysosomes

KW - TOR Serine-Threonine Kinases

KW - Journal Article

KW - Research Support, Non-U.S. Gov't

KW - Review

U2 - 10.1007/s00441-010-1115-0

DO - 10.1007/s00441-010-1115-0

M3 - SCORING: Review article

C2 - 21286756

VL - 343

SP - 467

EP - 473

JO - CELL TISSUE RES

JF - CELL TISSUE RES

SN - 0302-766X

IS - 3

ER -