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Epithelial-mesenchymal Transition: focus on metastatic cascade, alternative splicing, non-coding RNAs and modulating compounds

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Epithelial-mesenchymal Transition: focus on metastatic cascade, alternative splicing, non-coding RNAs and modulating compounds. / Samatov, Timur R; Tonevitsky, Alexander G; Schumacher, Udo.

in: MOL CANCER, Jahrgang 12, Nr. 1, 01.01.2013, S. 107.

Publikationen: SCORING: Beitrag in Fachzeitschrift/Zeitung › SCORING: Zeitschriftenaufsatz › Forschung › Begutachtung

Harvard

Samatov, TR, Tonevitsky, AG & Schumacher, U 2013, 'Epithelial-mesenchymal Transition: focus on metastatic cascade, alternative splicing, non-coding RNAs and modulating compounds', MOL CANCER, Jg. 12, Nr. 1, S. 107. https://doi.org/10.1186/1476-4598-12-107

APA

Samatov, T. R., Tonevitsky, A. G., & Schumacher, U. (2013). Epithelial-mesenchymal Transition: focus on metastatic cascade, alternative splicing, non-coding RNAs and modulating compounds. MOL CANCER, 12(1), 107. https://doi.org/10.1186/1476-4598-12-107

Vancouver

Samatov TR, Tonevitsky AG, Schumacher U. Epithelial-mesenchymal Transition: focus on metastatic cascade, alternative splicing, non-coding RNAs and modulating compounds. MOL CANCER. 2013 Jan 1;12(1):107. https://doi.org/10.1186/1476-4598-12-107

Bibtex

@article{bff39a0b3d30410fb4d6bc5397500109,
title = "Epithelial-mesenchymal Transition: focus on metastatic cascade, alternative splicing, non-coding RNAs and modulating compounds",
abstract = "Epithelial-mesenchymal transition (EMT) is a key process in embryonic development and metastases formation during malignant progression. This review focuses on transcriptional regulation, non-coding RNAs, alternative splicing events and cell adhesion molecules regulation during EMT. Additionally, we summarize the knowledge with regard to the small potentially druggable molecules capable of modulating EMT for cancer therapy.",
author = "Samatov, {Timur R} and Tonevitsky, {Alexander G} and Udo Schumacher",
year = "2013",
month = jan,
day = "1",
doi = "10.1186/1476-4598-12-107",
language = "English",
volume = "12",
pages = "107",
journal = "MOL CANCER",
issn = "1476-4598",
publisher = "BioMed Central Ltd.",
number = "1",

}

RIS

TY - JOUR

T1 - Epithelial-mesenchymal Transition: focus on metastatic cascade, alternative splicing, non-coding RNAs and modulating compounds

AU - Samatov, Timur R

AU - Tonevitsky, Alexander G

AU - Schumacher, Udo

PY - 2013/1/1

Y1 - 2013/1/1

N2 - Epithelial-mesenchymal transition (EMT) is a key process in embryonic development and metastases formation during malignant progression. This review focuses on transcriptional regulation, non-coding RNAs, alternative splicing events and cell adhesion molecules regulation during EMT. Additionally, we summarize the knowledge with regard to the small potentially druggable molecules capable of modulating EMT for cancer therapy.

AB - Epithelial-mesenchymal transition (EMT) is a key process in embryonic development and metastases formation during malignant progression. This review focuses on transcriptional regulation, non-coding RNAs, alternative splicing events and cell adhesion molecules regulation during EMT. Additionally, we summarize the knowledge with regard to the small potentially druggable molecules capable of modulating EMT for cancer therapy.

U2 - 10.1186/1476-4598-12-107

DO - 10.1186/1476-4598-12-107

M3 - SCORING: Journal article

C2 - 24053443

VL - 12

SP - 107

JO - MOL CANCER

JF - MOL CANCER

SN - 1476-4598

IS - 1

ER -