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Cross-talk between inflammation and coagulation

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Cross-talk between inflammation and coagulation. / Bickmann, Julia; Kiencke, Verena; Long, Andy; Renne, Thomas.

Hematology Education, 2017. 2017. S. 188 - 190.

Publikationen: SCORING: Beitrag in Buch/Sammelwerk › Konferenzbeitrag - Andere Beiträge zu Konferenzen › Lehre › Begutachtung

Harvard

Bickmann, J, Kiencke, V, Long, A & Renne, T 2017, Cross-talk between inflammation and coagulation. in Hematology Education, 2017. S. 188 - 190.

APA

Bickmann, J., Kiencke, V., Long, A., & Renne, T. (2017). Cross-talk between inflammation and coagulation. in Hematology Education, 2017 (S. 188 - 190)

Vancouver

Bickmann J, Kiencke V, Long A, Renne T. Cross-talk between inflammation and coagulation. in Hematology Education, 2017. 2017. S. 188 - 190

Bibtex

@inbook{abe68da0a35a45ab99b72277090191e5,
title = "Cross-talk between inflammation and coagulation",
abstract = "Take-home messages- Coagulation and inflammation are closely linked and the FXII-driven contact activation system is an example for this inti-mate crosstalk.- Platelet-released polyP forms nanoparticles that are retained on platelet surfaces where they directly initiate FXII activation..- A routine assay for platelet polyP exists and may be used to establish the polymer as a biomarker for thrombosis.- Targeting polyP protects from thrombosis in an FXII-dependent manner in vivo without elevating the bleeding risk.",
author = "Julia Bickmann and Verena Kiencke and Andy Long and Thomas Renne",
note = "javascript:void(0);",
year = "2017",
language = "Deutsch",
pages = "188 -- 190",
booktitle = "Hematology Education, 2017",

}

RIS

TY - CHAP

T1 - Cross-talk between inflammation and coagulation

AU - Bickmann, Julia

AU - Kiencke, Verena

AU - Long, Andy

AU - Renne, Thomas

N1 - javascript:void(0);

PY - 2017

Y1 - 2017

N2 - Take-home messages- Coagulation and inflammation are closely linked and the FXII-driven contact activation system is an example for this inti-mate crosstalk.- Platelet-released polyP forms nanoparticles that are retained on platelet surfaces where they directly initiate FXII activation..- A routine assay for platelet polyP exists and may be used to establish the polymer as a biomarker for thrombosis.- Targeting polyP protects from thrombosis in an FXII-dependent manner in vivo without elevating the bleeding risk.

AB - Take-home messages- Coagulation and inflammation are closely linked and the FXII-driven contact activation system is an example for this inti-mate crosstalk.- Platelet-released polyP forms nanoparticles that are retained on platelet surfaces where they directly initiate FXII activation..- A routine assay for platelet polyP exists and may be used to establish the polymer as a biomarker for thrombosis.- Targeting polyP protects from thrombosis in an FXII-dependent manner in vivo without elevating the bleeding risk.

M3 - Konferenzbeitrag - Andere Beiträge zu Konferenzen

SP - 188

EP - 190

BT - Hematology Education, 2017

ER -