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Coronary plaque injury triggers neutrophil activation in patients with coronary artery disease.

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Coronary plaque injury triggers neutrophil activation in patients with coronary artery disease. / Rudolph, Volker; Steven, Daniel; Gehling, Ursula; Goldmann, Britta; Rudolph, Tanja Katharina; Friedrichs, Kai; Meinertz, Thomas; Heitzer, Thomas; Baldus, Stephan.

in: FREE RADICAL BIO MED, Jahrgang 42, Nr. 4, 4, 2007, S. 460-465.

Publikationen: SCORING: Beitrag in Fachzeitschrift/Zeitung › SCORING: Zeitschriftenaufsatz › Forschung › Begutachtung

Harvard

Rudolph, V, Steven, D, Gehling, U, Goldmann, B, Rudolph, TK, Friedrichs, K, Meinertz, T, Heitzer, T & Baldus, S 2007, 'Coronary plaque injury triggers neutrophil activation in patients with coronary artery disease.', FREE RADICAL BIO MED, Jg. 42, Nr. 4, 4, S. 460-465. <http://www.ncbi.nlm.nih.gov/pubmed/17275677?dopt=Citation>

APA

Rudolph, V., Steven, D., Gehling, U., Goldmann, B., Rudolph, T. K., Friedrichs, K., Meinertz, T., Heitzer, T., & Baldus, S. (2007). Coronary plaque injury triggers neutrophil activation in patients with coronary artery disease. FREE RADICAL BIO MED, 42(4), 460-465. [4]. http://www.ncbi.nlm.nih.gov/pubmed/17275677?dopt=Citation

Vancouver

Rudolph V, Steven D, Gehling U, Goldmann B, Rudolph TK, Friedrichs K et al. Coronary plaque injury triggers neutrophil activation in patients with coronary artery disease. FREE RADICAL BIO MED. 2007;42(4):460-465. 4.

Bibtex

@article{e5d0eb4c27294e32b2c1923ef5e27c74,
title = "Coronary plaque injury triggers neutrophil activation in patients with coronary artery disease.",
abstract = "Activation of leukocytes, in particular polymorphonuclear neutrophils (PMN), is considered an early event in unstable coronary disease. Upon activation PMN liberate myeloperoxidase (MPO), an enzyme which binds to the vessel wall and depletes vascular NO bioavailability. Using coronary balloon angioplasty as a trigger to provoke coronary plaque injury, we assessed the time course of neutrophil activation, local and peripheral levels of myeloperoxidase, and systemic vascular NO bioavailability in patients with stable coronary artery disease. Twenty-four patients with stable CAD were enrolled prior to undergoing percutaneous interventions (PCI, n=14) and diagnostic coronary angiography (n=10), respectively. Following angioplasty arterial MPO plasma levels increased (231.5+/-67.6 to 273.8+/-80.4 pg/mg protein; P",
author = "Volker Rudolph and Daniel Steven and Ursula Gehling and Britta Goldmann and Rudolph, {Tanja Katharina} and Kai Friedrichs and Thomas Meinertz and Thomas Heitzer and Stephan Baldus",
year = "2007",
language = "Deutsch",
volume = "42",
pages = "460--465",
journal = "FREE RADICAL BIO MED",
issn = "0891-5849",
publisher = "Elsevier Inc.",
number = "4",

}

RIS

TY - JOUR

T1 - Coronary plaque injury triggers neutrophil activation in patients with coronary artery disease.

AU - Rudolph, Volker

AU - Steven, Daniel

AU - Gehling, Ursula

AU - Goldmann, Britta

AU - Rudolph, Tanja Katharina

AU - Friedrichs, Kai

AU - Meinertz, Thomas

AU - Heitzer, Thomas

AU - Baldus, Stephan

PY - 2007

Y1 - 2007

N2 - Activation of leukocytes, in particular polymorphonuclear neutrophils (PMN), is considered an early event in unstable coronary disease. Upon activation PMN liberate myeloperoxidase (MPO), an enzyme which binds to the vessel wall and depletes vascular NO bioavailability. Using coronary balloon angioplasty as a trigger to provoke coronary plaque injury, we assessed the time course of neutrophil activation, local and peripheral levels of myeloperoxidase, and systemic vascular NO bioavailability in patients with stable coronary artery disease. Twenty-four patients with stable CAD were enrolled prior to undergoing percutaneous interventions (PCI, n=14) and diagnostic coronary angiography (n=10), respectively. Following angioplasty arterial MPO plasma levels increased (231.5+/-67.6 to 273.8+/-80.4 pg/mg protein; P

AB - Activation of leukocytes, in particular polymorphonuclear neutrophils (PMN), is considered an early event in unstable coronary disease. Upon activation PMN liberate myeloperoxidase (MPO), an enzyme which binds to the vessel wall and depletes vascular NO bioavailability. Using coronary balloon angioplasty as a trigger to provoke coronary plaque injury, we assessed the time course of neutrophil activation, local and peripheral levels of myeloperoxidase, and systemic vascular NO bioavailability in patients with stable coronary artery disease. Twenty-four patients with stable CAD were enrolled prior to undergoing percutaneous interventions (PCI, n=14) and diagnostic coronary angiography (n=10), respectively. Following angioplasty arterial MPO plasma levels increased (231.5+/-67.6 to 273.8+/-80.4 pg/mg protein; P

M3 - SCORING: Zeitschriftenaufsatz

VL - 42

SP - 460

EP - 465

JO - FREE RADICAL BIO MED

JF - FREE RADICAL BIO MED

SN - 0891-5849

IS - 4

M1 - 4

ER -