PortalPublikationenAutonomous innervation in renal inflammatory disease-innocen...

Autonomous innervation in renal inflammatory disease-innocent bystander or active modulator?

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Autonomous innervation in renal inflammatory disease-innocent bystander or active modulator? / Ditting, Tilmann; Tiegs, Gisa; Veelken, Roland.

in: J MOL MED, Jahrgang 87, Nr. 9, 9, 2009, S. 865-870.

Publikationen: SCORING: Beitrag in Fachzeitschrift/Zeitung › SCORING: Zeitschriftenaufsatz › Forschung › Begutachtung

Harvard

Ditting, T, Tiegs, G & Veelken, R 2009, 'Autonomous innervation in renal inflammatory disease-innocent bystander or active modulator?', J MOL MED, Jg. 87, Nr. 9, 9, S. 865-870. <http://www.ncbi.nlm.nih.gov/pubmed/19597724?dopt=Citation>

APA

Ditting, T., Tiegs, G., & Veelken, R. (2009). Autonomous innervation in renal inflammatory disease-innocent bystander or active modulator? J MOL MED, 87(9), 865-870. [9]. http://www.ncbi.nlm.nih.gov/pubmed/19597724?dopt=Citation

Vancouver

Ditting T, Tiegs G, Veelken R. Autonomous innervation in renal inflammatory disease-innocent bystander or active modulator? J MOL MED. 2009;87(9):865-870. 9.

Bibtex

@article{7bc589deeb0c491ca2791a3a12fe15fd,
title = "Autonomous innervation in renal inflammatory disease-innocent bystander or active modulator?",
abstract = "Increasing evidence suggests a significant interrelation between the autonomic nervous system and the immune system. The kidney is innervated by efferent sympathetic nerves as well as by peptidergic sensory afferent nerve fibers. Inflammation in the kidney may be affected by both types of fibers. Peptidergic {"}sensory{"} neurons might play a particularly important role: These fibers can induce local neurogenic inflammation via paracrine effects of their transmitters and evoke increased efferent sympathetic nerve outflow via their projections to the central nervous system. Several reports support the notion that renal innervation does indeed contribute to inflammation and sclerosis in kidney diseases. Hence, receptor antagonists interfering with the interaction of innervation and the immune system may prove useful to mitigate inflammatory processes in the kidney.",
author = "Tilmann Ditting and Gisa Tiegs and Roland Veelken",
year = "2009",
language = "Deutsch",
volume = "87",
pages = "865--870",
journal = "J MOL MED",
issn = "0946-2716",
publisher = "Springer",
number = "9",

}

RIS

TY - JOUR

T1 - Autonomous innervation in renal inflammatory disease-innocent bystander or active modulator?

AU - Ditting, Tilmann

AU - Tiegs, Gisa

AU - Veelken, Roland

PY - 2009

Y1 - 2009

N2 - Increasing evidence suggests a significant interrelation between the autonomic nervous system and the immune system. The kidney is innervated by efferent sympathetic nerves as well as by peptidergic sensory afferent nerve fibers. Inflammation in the kidney may be affected by both types of fibers. Peptidergic "sensory" neurons might play a particularly important role: These fibers can induce local neurogenic inflammation via paracrine effects of their transmitters and evoke increased efferent sympathetic nerve outflow via their projections to the central nervous system. Several reports support the notion that renal innervation does indeed contribute to inflammation and sclerosis in kidney diseases. Hence, receptor antagonists interfering with the interaction of innervation and the immune system may prove useful to mitigate inflammatory processes in the kidney.

AB - Increasing evidence suggests a significant interrelation between the autonomic nervous system and the immune system. The kidney is innervated by efferent sympathetic nerves as well as by peptidergic sensory afferent nerve fibers. Inflammation in the kidney may be affected by both types of fibers. Peptidergic "sensory" neurons might play a particularly important role: These fibers can induce local neurogenic inflammation via paracrine effects of their transmitters and evoke increased efferent sympathetic nerve outflow via their projections to the central nervous system. Several reports support the notion that renal innervation does indeed contribute to inflammation and sclerosis in kidney diseases. Hence, receptor antagonists interfering with the interaction of innervation and the immune system may prove useful to mitigate inflammatory processes in the kidney.

M3 - SCORING: Zeitschriftenaufsatz

VL - 87

SP - 865

EP - 870

JO - J MOL MED

JF - J MOL MED

SN - 0946-2716

IS - 9

M1 - 9

ER -